- ►tno.co.jp [PDF] AM van den Hoek, AC Heijboer, EPM Corssmit, … - Diabetes, 2004 - Am Diabetes Assoc Peptide YY 3–36 (PYY 3–36 ) is released by the gut in response to nutrient
ingestion. It modulates the activities of orexigenic neuropeptide Y (NPY)
neurons and anorexigenic proopiomelanocortin (POMC) neurons in the ... Cited by 41 - Related articles - BL Direct - All 5 versions
- ►tno.co.jp [PDF] AM van den Hoek, PJ Voshol, BN Karnekamp, … - Diabetes, 2004 - Am Diabetes Assoc Recent evidence demonstrates that hypothalamic insulin signaling is required for
inhibition of endogenous glucose production. The downstream mechanisms that are
responsible for the effects of hypothalamic insulin receptor activation on ... Cited by 31 - Related articles - BL Direct - All 7 versions
AM van den Hoek, AC Heijboer, EPM Corssmit, … - DIABETES, 2004 - Am Diabetes Assoc Peptide YY 3–36 (PYY 3–36 ) is released by the gut in re- sponse to nutrient
ingestion. It modulates the activities of orexigenic neuropeptide Y (NPY)
neurons and an- orexigenic proopiomelanocortin (POMC) neurons in the ... Related articles
F Hoentjen, WPM Hopman, J Jansen - Scandinavian journal of gastroenterology, 2001 - informahealthcare.com PYY is a gastrointestinal peptide, rst isolated from the ... PYY on
meal-stimulated gallbladder emptying is not known. Because PYY is a potent
inhibitor of the cephalic phase of gastric acid secretion in dogs (21, 22) ... Cited by 33 - Related articles - BL Direct - All 7 versions
- ►diabetesjournals.org PJ Voshol, MC Jong, VEH Dahlmans, D Kratky … - Diabetes, 2001 - Am Diabetes Assoc In patients with type 2 diabetes, a strong correlation between accumulation of
intramuscular triclycerides (TGs) and insulin resistance has been found. The aim
of the present study was to determine whether there is a causal relation ... Cited by 66 - Related articles - BL Direct - All 9 versions
BG Challis, SB Pinnock, AP Coll, RN Carter, SL … - Biochemical and biophysical research communications, 2003 - Elsevier It has recently been suggested that gut-derived PYY 3–36 may be involved in
the central mediation of post-prandial satiety signals. We have examined the
acute effects of peripherally administered PYY 3–36 on food intake and ... Cited by 124 - Related articles - All 5 versions
- ►tno.co.jp [PDF] AC Heijboer, AM van den Hoek, H Pijl, PJ … - Diabetologia, 2005 - Springer Abstract Aims/hypothesis: The present study was con- ducted to evaluate the
effects of central administration of melanotan II (MTII), a melanocortin-3/4
receptor agonist, on hepatic and whole-body insulin sensitivity, ... Cited by 13 - Related articles - BL Direct - All 4 versions
- ►nutrition.org SH Adams, C Lei, CM Jodka, SE Nikoulina, JA … - Journal of Nutrition, 2006 - Am Soc Nutrition In rodents, weight reduction after peptide YY[3-36] (PYY[3-36]) administration
may be due largely to decreased food consumption. Effects on other processes
affecting energy balance (energy expenditure, fuel partitioning, gut ... Cited by 37 - Related articles - BL Direct - All 10 versions
RA Pittner, CX Moore, SP Bhavsar, BR … - International Journal of Obesity, 2004 - cababstractsplus.org BACKGROUND: Peptide YY (PYY) is a 36 amino-acid peptide secreted from ileal L
cells following meals. The cleaved subpeptide PYY [3-36] is biologically active
and may constitute the majority of circulating PYY-like immunoreactivity. ... Cited by 117 - Related articles - All 3 versions
JL Marks, K Waite - Journal of neuroendocrinology, 1997 - interscience.wiley.com Acute administration of neuropeptide Y(NPY) into the hypothalamus and third
cerebral ventricle (ICV) increases respiratory quotient, reduces energy
expenditure and increases circulating, insulin, glucagon and ... Cited by 26 - Related articles - BL Direct - All 5 versions
