AI Duarte, MS Santos, R Seiça, CR Oliveira - Diabetes, 2004 - Am Diabetes Assoc Evidence suggests that oxidative stress is involved in the pathophysiology of diabetic complications
and that insulin has a neuroprotective role in oxidative stress conditions. In this study, we evaluated
the in vitro effect of insulin in the susceptibility to oxidative stress and in the transport of ... Cited by 12 - Related articles - BL Direct - All 4 versions
A Duarte, M Santos, R Seiça, C Resende de … - …, 2000 - content.karger.com It has been suggested that increased oxidative stress might be involved in the pathophysiology
of diabetic complications. In this study, we investigated the effect of diabetes on the susceptibility
of synaptosomes to oxidative stress (induced by the oxidizing pair ascorbate/Fe 2+ ) and ... Cited by 7 - Related articles - BL Direct - All 5 versions
- ►uc.pt [PDF] AI Duarte, MS Santos, R Seiça, CR Oliveira - Brain research, 2003 - Elsevier In this study, we investigated the in vitro effect of exogenously administered insulin on the susceptibility
to oxidative stress and on the accumulation of the amino acid neurotransmitters γ-aminobutyric
acid (GABA) and glutamate in a synaptosomal fraction isolated from male Wistar rat brain ... Cited by 7 - Related articles - All 6 versions
J Chen, CG Östenson - Pancreas, 2005 - journals.lww.com Skip Navigation Links Home > May 2005 - Volume 30 - Issue 4 > Inhibition of Protein-tyrosine
Phosphatases Stimulates Insul... ... From the Department of Molecular Medicine, Endocrine and
Diabetes Unit, Karolinska Hospital, Stockholm, Sweden. ... Supported by grants from the ... Cited by 5 - Related articles - All 4 versions
BM Lewis, IS Ismail, B Issa, JR Peters, MF … - Journal of …, 1996 - Soc Endocrinology We have studied the effects of glucose on the release of somatostatin (SS), TRH and GHRH
from incubated hypothalami of normal and genetically diabetic, Goto-Kakizaki (GK) rats. The
active isomer D-glucose caused a dose-related inhibition of SS, TRH and GHRH from ... Cited by 5 - Related articles - BL Direct - All 7 versions
MS Santos, AI Duarte, MJ Matos, T Proença, R Seiça, … - Life Sciences, 2000 - Elsevier Increased oxidative stress is believed to be an important factor in the development of diabetic
complications. In this study, the effect of diabetes on the susceptibility of synaptosomes to oxidative
stress, induced by the oxidizing system ascorbate/Fe 2 +, on the activity of antioxidant ... Cited by 6 - Related articles - BL Direct - All 3 versions
- ►endojournals.org H Mosen, A Salehi, P Alm, R Henningsson, J Jimenez- … - Endocrinology, 2005 - Endocrine Soc The Goto-Kakizaki (GK) rat displays a markedly reduced insulin response to glucose, a defect
that is thought to be coupled to an impaired glucose signaling in the ß-cell. We have examined
whether carbon monoxide (CO), derived from ß-cell heme oxygenase (HO), might be ... Cited by 10 - Related articles - All 4 versions
- ►diabetesjournals.org M Dolz, D Bailbé, MH Giroix, S Calderari, MN … - Diabetes, 2005 - Am Diabetes Assoc Because acetylcholine (ACh) is a recognized potentiator of glucose-stimulated insulin release
in the normal β-cell, we have studied ACh's effect on islets of the Goto-Kakizaki (GK) rat, a spontaneous
model of type 2 diabetes. We first verified that ACh was able to restore the insulin ... Cited by 10 - Related articles - BL Direct - All 6 versions
[CITATION] Cyclic AMP regulation of lactate dehydrogenase
A Guenifi, E Simonsson, S Karlsson, B Ahrén, SM … - Pancreas, 2001 - journals.lww.com Page 1. Carbachol Restores Insulin Release in Diabetic GK Rat Islets by Mechanisms Largely
Involving Hydrolysis of Diacylglycerol and Direct Interaction with the Exocytotic Machinery Amel
Guenifi, *Erik Simonsson, *Sven Karlsson, *Bo Ahrén, and Samy M. Abdel-Halim ... Cited by 11 - Related articles - BL Direct - All 3 versions
