related:http://diabetes.diabetesjournals.org/cgi/content/abstract/53/9/2261

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►Increased serum leptin protects from adiposity despite the increased glucose …

Y Terauchi, J Matsui, J Kamon, T Yamauchi, N Kubota, … - Diabetes, 2004 - Am Diabetes Assoc
Mice lacking the p85α regulatory subunit of phosphoinositide (PI) 3-kinase (Pik3r1 −/− ) showed
increased glucose uptake in white adipose tissue (WAT) and skeletal muscle due to increased
phosphatidylinositol (3,4,5)-triphosphate [PtdIns(3,4,5)P3] production and on a normal ...
Cited by 3 - Related articles - All 3 versions

[PDF] ►Increased serum leptin protects from adiposity despite the increased glucose …

Y Terauchi, J Matsui, J Kamon, T Yamauchi, … - DIABETES-NEW …, 2004 - Am Diabetes Assoc
Page 1. Increased Serum Leptin Protects From Adiposity Despite the Increased Glucose Uptake
in White Adipose Tissue in Mice Lacking p85 Phosphoinositide 3-Kinase Yasuo Terauchi, 1,2
Junji Matsui, 1 Junji Kamon, 1 Toshimasa Yamauchi, 1,2 Naoto Kubota, 1,2,3 ...
Cited by 8 - Related articles - BL Direct - All 7 versions

[CITATION] Erickson MRS, Yen FT, Bihain BE, Lodish HF: Proteolytic cleavage product of 30- …

J Fruebis, TS Tsao, S Javorschi, D Ebbets-Reed, RS … - Proc Natl Acad Sci USA, 2001
Cited by 2 - Related articles

[CITATION] Low circulating adiponectin/Acrp30 levels in a mouse model of acromegaly

JP del Rincon, P Hellmann, KT Coschigano, JJ … - the Endocrine Society 85th …, 2003
Cited by 4 - Related articles

[CITATION] Transgenic mice overexpressing growth hormone are insulin resistant due to …

T Higashimori, HJ Kim, K Coschigano, SY Park, H Choi … - Diabetes (Suppl. 1), 2003
Cited by 4 - Related articles

[CITATION] Replenishment of the fat-derived hormone adiponectin reverses insulin resistance …

T Yamauchi, J Kamon, H Waki, Y Terauchi, N Kubota, K … - Nat. Med, 2001
Cited by 7 - Related articles

►The p85 $# x003b1; Regulatory Subunit of Phosphoinositide 3-Kinase Potentiates …

CM Taniguchi… - Molecular and Cellular Biology, 2007 - ukpmc.ac.uk
Insulin resistance is a defining feature of type 2 diabetes and the metabolic syndrome. While
the molecular mechanisms of insulin resistance are multiple, recent evidence suggests that attenuation
of insulin signaling by c-Jun N-terminal kinase (JNK) may be a central part of the ...
Related articles

►N-terminal domains of the class ia phosphoinositide 3-kinase regulatory subunit …

KM Hill, Y Huang, SC Yip, J Yu, JE Segall, JM … - Journal of Biological …, 2001 - ASBMB
Phosphoinositide (PI) 3-kinases are required for the acute regulation of the cytoskeleton by growth
factors. We have shown previously that in the MTLn3 rat adenocarcinoma cells line, the
p85/p110α PI 3-kinase is required for epidermal growth factor (EGF)-stimulated ...
Cited by 21 - Related articles - BL Direct - All 3 versions

The p85 {alpha} regulatory subunit of phosphoinositide 3-kinase potentiates c-Jun …

- ►nih.gov
CM Taniguchi, JO Aleman, K Ueki, J Luo, T … - Molecular and Cellular …, 2007 - Am Soc Microbiol
Insulin resistance is a defining feature of type 2 diabetes and the metabolic syndrome. While
the molecular mechanisms of insulin resistance are multiple, recent evidence suggests that attenuation
of insulin signaling by c-Jun N-terminal kinase (JNK) may be a central part of the ...
Cited by 10 - Related articles - BL Direct - All 7 versions

Reduced glucose incorporation to triglycerides following chronic exposure of …

G Nyberg, S Bostrom, R Johansson, U Smith - Acta Endocrinologica, 1980 - eje.org
Using the tissue culture technique we have recently demonstrated that long-term exposure of
human adipose tissue to human growth hormone (GH) in vitro leads to an impairment in glucose
incorporation into triglycerides. This effect was further studied in the present investigation. ...
Cited by 10 - Related articles - All 6 versions

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