TJ Biden, D Robinson, D Cordery, WE Hughes, AK … - Diabetes, 2004 - Am Diabetes Assoc Type 2 diabetes can be viewed as a failure of the pancreatic β-cell to compensate for peripheral
insulin resistance with enhanced insulin secretion. This failure is explained by both a relative
loss of β-cell mass as well as secretory defects that include enhanced basal secretion ... Cited by 18 - Related articles - BL Direct - All 4 versions
- ►nih.gov A Podcheko, P Northcott, G Bikopoulos, A Lee … - Molecular and Cellular …, 2007 - Am Soc Microbiol The pleckstrin homology domain-interacting protein (PHIP) was originally identified as a
902-amino-acid (aa) protein that regulates insulin receptor-stimulated GLUT4 translocation in
skeletal-muscle cells. Immunoblotting and immunohistological analyses of pancreatic ... Cited by 3 - Related articles - BL Direct - All 6 versions
H Zhang, ZH Li, MQ Zhang, MS Katz, BX Zhang - Journal of Biological …, 2008 - ASBMB Nonesterified fatty acids may influence mitochondrial function by alterations in gene
expression, metabolism, and/or mitochondrial Ca 2+ ([Ca 2+ ] m ) homeostasis. We have previously
reported that polyunsaturated fatty acids induce Ca 2+ efflux from mitochondria, an action ... Cited by 2 - Related articles - All 3 versions
- ►physiology.org [PDF] W Zhang, H Meng, ZH Li, Z Shu, X Ma, … - American Journal of …, 2007 - Am Physiological Soc 52 articles, 26 of which you can access free at: This article cites http://ajprenal.physiology.org
/cgi/content/full/292/3/F1054#BIBL ... 3 other HighWire hosted articles: This article has been
cited by ... [PDF] [Full Text] [Abstract] , December 1, 2007; 293 (6): F1768-F1776. Am J ... Cited by 5 - Related articles - BL Direct - All 7 versions
- ►physiology.org BX Zhang, X Ma, W Zhang, CK Yeh, A Lin, … - American Journal of …, 2006 - Am Physiological Soc In a variety of disorders, overaccumulation of lipid in nonadipose tissues, including the heart,
skeletal muscle, kidney, and liver, is associated with deterioration of normal organ function, and
is accompanied by excessive plasma and cellular levels of free fatty acids (FA). Increased ... Cited by 6 - Related articles - BL Direct - All 3 versions
- ►nih.gov MS Parihar, RR Nazarewicz, E Kincaid, U Bringold, … - … and biophysical research …, 2008 - Elsevier Mitochondrial nitric oxide (NO) synthase (mtNOS) regulates various functions of mitochondria
in liver [1], [2], [3], [4] and [5], heart [6], [7] and [8], brain [2], [9] and [10], kidney [11] and [12], as
well as cells including endothelial [13], kidney [14], neuronal [15], cardiac myocytes [16], ... Cited by 6 - Related articles - All 5 versions
L Yi, J He, Y Liang, D Yuan, H Gao, H Zhou - Chemistry and Physics of Lipids, 2007 - Elsevier Fatty acids, having intimate relationship with type 2 diabetes mellitus (DM2), are not only the
main energy source as nutrients, but also signaling molecules in insulin secretion. In this
work, we developed a two-step rapid method to comprehensive profiling of esterified fatty ... Cited by 8 - Related articles - All 3 versions
- ►nih.gov J Farhang-Fallah, VK Randhawa, A Nimnual, … - Molecular and Cellular …, 2002 - Am Soc Microbiol Receptor-mediated tyrosine phosphorylation of the insulin receptor substrate 1 (IRS-1) is required
for the propagation of many of insulin's biological effects. The amino-terminal pleckstrin homology
(PH) domain of IRS-1 plays a pivotal role in promoting insulin receptor (IR)-IRS-1 protein ... Cited by 20 - Related articles - BL Direct - All 5 versions
H Duplain, C Sartori, P Dessen, PY Jayet, … - The Journal of …, 2008 - pubmedcentral.nih.gov Peroxynitrite synthesis is increased in insulin resistant animals and humans. Peroxynitirite-induced
nitration of insulin signalling proteins impairs insulin action in vitro, but the role of peroxynitrite
in the pathogenesis of insulin resistance in vivo is not known. We therefore assessed the ... Cited by 8 - Related articles - All 6 versions
JN Sheu, TH Lin, CK Lii, CC Chen, HW Chen, KL … - Food and Chemical …, 2006 - Elsevier That both infiltrating macrophages and resident mesangial cells express inducible nitric oxide
synthase (iNOS) and produce nitric oxide (NO) excessively is crucial to the progress of
glomerulonephritis. Although several reports have mentioned the protective impacts of ... Cited by 10 - Related articles - All 6 versions
