- ►diabetesjournals.org ME Røder, SE Kahn - Diabetes, 2004 - Am Diabetes Assoc Disproportionate hyperproinsulinemia is a feature of β-cell dysfunction in type 2 diabetes. It has
been hypothesized that this abnormality represents an intrinsic abnormality of the β-cell
and/or may result from an increase in β-cell secretory demand. To address this, six ... Cited by 2 - Related articles - BL Direct - All 5 versions
M Faehling, RD Fussgaenger, PM Jehle - Diabetologia, 1999 - ncbi.nlm.nih.gov 1: Diabetologia. 1999 Feb;42(2):259-60. Comment on: Diabetologia. 1998
Mar;41(3):249-56. High affinity binding sites for proinsulin on human umbilical vein
endothelial cells (HUVEC). Faehling M, Fussgaenger RD, Jehle PM. ... Cited by 3 - Related articles - BL Direct - All 2 versions
CM MacNamara, BA Barrow, SE Manley, JC Levy, … - Diabetes research and …, 2000 - Elsevier Elevated proinsulin secretion and islet amyloid deposition are both features of Type 2 diabetes
but their relationship to β-cell dysfunction is unknown. To determine if islet amyloid polypeptide
(IAPP) secretion is disproportionate with other β-cell products at any stage of glucose ... Cited by 3 - Related articles - All 6 versions
[CITATION] Le role de l'obesité dans l'etiopathogenie du diabete sucre
C Ionescu-Tîrgovişte, D Cheţa, MI Elena Popa - Medecine et Nutrition, 1976 Cited by 3 - Related articles
[CITATION] The Belgian Diabetes Registry: Proinsulin levels and the proinsulin C-peptide ratio …
C Ionescu - Diabetic Medicine - interscience.wiley.com An analysis of the last 20 000 newly diagnosed diabetic patients consecutively registered from
1 January 1981 to 6 June 1991 in the Bucharest Registry of Diabetes showed the following:
(1) primary insulin-dependence (Type 1 diabetes) was encountered in only 7% of cases: ... Cited by 4 - Related articles - BL Direct - All 4 versions
C Guja, L GUJA, S NUTLAND, H RANCE, JA Todd … - Revue roumaine de …, 2004 - cat.inist.fr Type 1 diabetes(T 1 DM) is a common, chronic disease with autoimmune pathogeny, conditioned
by genetic factors. Class II HLA DR and DQ and insulin gene polymorphisms encode for most
of the T 1 DM genetic susceptibility. We have previously shown that class I alleles of the ... Cited by 4 - Related articles - All 2 versions
