GT Mustata, M Rosca, KM Biemel, O Reihl, MA … - Diabetes, 2005 - Am Diabetes Assoc We tested the hypothesis that green tea prevents diabetes-related tissue
dysfunctions attributable to oxidation. Diabetic rats were treated daily with
tap water, vitamins C and E, or fresh Japanese green tea extract. After 12 ... Cited by 31 - Related articles - All 7 versions
I Retinopathy, RM Defects - DIABETES, 2005 - Am Diabetes Assoc Page 1. Paradoxical Effects of Green Tea (Camellia Sinensis) and Antioxidant Vitamins
in Diabetic Rats Improved Retinopathy and Renal Mitochondrial Defects ... Related articles
PVA Babu, KE Sabitha, CS Shyamaladevi - Chemico-biological interactions, 2006 - Elsevier Diabetes-induced hyperlipidemia, oxidative stress and protein glycation impair
cellular calcium and sodium homeostasis associated with abnormal membrane-bound
enzyme activities resulting in cardiac dysfunction in diabetes. To explore ... Cited by 11 - Related articles - All 5 versions
SM Culbertson, EI Vassilenko, LD Morrison, K … - Journal of Biological Chemistry, 2003 - ASBMB The inhibition of post-Amadori advanced glycation end product (AGE) formation by
three different classes of AGE inhibitors, carbonyl group traps, chelators, and
radical-trapping antioxidants, challenge the current paradigms that: 1) AGE ... Cited by 27 - Related articles - BL Direct - All 5 versions
RA Kowluru, V Kowluru, Y Xiong, YS Ho - Free Radical Biology and Medicine, 2006 - Elsevier The retina experiences mitochondrial dysfunction in diabetes, superoxide levels
are elevated, and mitochondrial superoxide dismutase (MnSOD) activity is
decreased. Inhibition of superoxide accumulation in diabetes prevents ... Cited by 40 - Related articles - All 5 versions
MF McCarty - Medical hypotheses, 2005 - Elsevier Increased endogenous generation of advanced glycation endproducts (AGEs)
contributes importantly to the vascular complications of diabetes, in part owing
to activation of the pro-inflammatory RAGE receptor. However, AGE-altered ... Cited by 19 - Related articles - All 19 versions
- ►iovs.org M Kanwar, PS Chan, TS Kern, RA Kowluru - Investigative ophthalmology & visual science, 2007 - ARVO RESULTS. Retinal mitochondria had twofold increase in superoxide levels in
nontransgenic (wild-type [WT]) diabetic mice compared with WT nondiabetic mice.
In the same retina, diabetes decreased mitochondrial GSH levels by 40% and ... Cited by 34 - Related articles - BL Direct - All 3 versions
L Pari, S Venkateswaran - Brazilian Journal of Medical and Biological Research, 2003 - SciELO Brasil Changes in the structural and functional properties of collagen caused by
advanced glycation might be of importance for the etiology of late complications
in diabetes. The present study was undertaken to investigate the influence ... Cited by 7 - Related articles - Cached - BL Direct - All 10 versions
JM Forbes, G Soldatos, MC Thomas - The Clinical biochemist. Reviews/Australian Association …, 2005 - pubmedcentral.nih.gov The contents of articles or advertisements in The Clinical Biochemist –
Reviews are not to be construed as official statements, evaluations or
endorsements by the AACB, its official bodies or its agents. Statements of ... Cited by 7 - Related articles - BL Direct - All 7 versions
- ►iovs.org RA Kowluru, L Atasi, YS Ho - Investigative ophthalmology & visual science, 2006 - ARVO PURPOSE. Apoptosis of retinal capillary cells is an early event in the
pathogenesis of retinopathy in diabetes, and oxidative stress has been linked to
accelerated apoptosis of retinal capillary cells. Mitochondria are the ... Cited by 34 - Related articles - BL Direct - All 5 versions
