FS Wong, W Du, IJ Thomas, L Wen - Diabetes, 2005 - Am Diabetes Assoc The most important genetic susceptibility factor for type 1 diabetes is encoded
in the major histocompatibility complex (MHC). The nonobese diabetic (NOD)
mouse, which develops spontaneous diabetes, expresses H-2 g7 comprising the ... Cited by 3 - Related articles - All 5 versions
- ►jimmunol.org A Ueno, S Cho, L Cheng, Z Wang, B Wang, Y … - The Journal of Immunology, 2005 - Am Assoc Immnol Polymorphism of MHC and MHC-linked genes is tightly associated with
susceptibility to type 1 diabetes (T1D) in human and animal models. Despite the
extensive studies, however, the role of MHC and MHC-linked genes expressed ... Cited by 3 - Related articles - BL Direct - All 5 versions
LS Mansfield, SR Abner - … Foodborne Diseases: Mechanisms of Pathogenesis and …, 2000 - books.google.com CHAPTER 6 Molecular Mechanisms Governing Campylobacter Pathogenicity LS
MANSFIELD SR ABNER 1. INTRODUCTION 1.1. SIGNIFICANCE OF CAMPYLOBACTER INFECTION
DISEASE in humans caused by Campylobacter spp. is a serious emerging ... Cited by 3 - Related articles
JD Carter, M Chen, JD Ellett, KM Smith, JL … - Transplantation proceedings, 2004 - Elsevier Autoimmune-mediated cytotoxicity may cause pancreatic islet transplant failure,
leading to recurrent diabetes. Protection of islet grafts depends on
immunosuppressive control, which may also prevent autoimmune recurrence of ... Cited by 3 - Related articles - All 16 versions
LS Mansfield, JS Patterson, BR Fierro, AJ … - Microbial Pathogenesis, 2008 - Elsevier We hypothesized that particular genetic backgrounds enhance rates of
colonization, increase severity of enteritis, and allow for extraintestinal
spread when inbred IL-10 −/− mice are infected with pathogenic C. ... Cited by 4 - Related articles - All 2 versions
[CITATION] Differential effects of anti-B7-B1 and anti-B7-B2 monoclonal antibody treatment on the …
Y Yang, P Santamaria - Journal of Autoimmunity, 2004 - Elsevier Insulin-dependent diabetes mellitus (Type 1 Diabetes, T1D) in humans and
nonobese diabetic (NOD) mice is the result of a chronic autoimmune process
directed against the pancreatic beta cells [1 and 2]. There is extensive ... Cited by 4 - Related articles - All 3 versions
N Klöting, I Klöting - Molecular genetics and metabolism, 2004 - Elsevier Spontaneous diabetes in B(io)B(reeding) rats is complex, polygenic, and
recessively inherited. Several crossing studies have demonstrated that beside
the class II genes of the major histocompatibility complex (MHC, Iddm1) ... Cited by 5 - Related articles - All 4 versions
- ►diabetesjournals.org EJ Gordon, LS Wicker, LB Peterson, DV … - Diabetes, 2005 - Am Diabetes Assoc Costimulation blockade induces prolonged rat islet and skin xenograft survival
in C57BL/6 mice. Nonobese diabetic (NOD) mice, which are used to model human
autoimmune diabetes, are resistant to costimulation blockade-induced ... Cited by 6 - Related articles - All 7 versions
BA Woda, ES Handler, DL Greiner, C … - Diabetes, 1991 - Am Diabetes Assoc Diabetes-prone (DP) BB rats develop spontaneous autoimmune insulin-dependent
diabetes mellitus (IDDM). The cell populations involved in the expression of
diabetes are not precisely known but probably include natural killer (NK) ... Cited by 6 - Related articles - All 4 versions
