- ►diabetesjournals.org M Jackerott, A Moldrup, P Thams, ED … - Diabetes, 2006 - Am Diabetes Assoc Pancreatic ß-cell growth and survival and insulin production are stimulated by
growth hormone and prolactin through activation of the transcription factor
signal transducer and activator of transcription (STAT)5. To assess the ... Cited by 13 - Related articles - BL Direct - All 5 versions
- ►endocrinology-journals.org AJ Weinhaus, LE Stout, NV Bhagroo, TC … - Journal of Endocrinology, 2007 - Soc Endocrinology Glucokinase activity is increased in pancreatic islets during pregnancy and in
vitro by prolactin (PRL). The underlying mechanisms that lead to increased
glucokinase have not been resolved. Since glucose itself regulates ... Cited by 14 - Related articles - BL Direct - All 3 versions
- ►nih.gov JY Lee, O Gavrilova, B Davani, R Na, GW … - BBA-Molecular Cell Research, 2007 - Elsevier In insulinoma cell lines proliferation and insulin gene transcription are
stimulated by growth hormone and prolactin, which convey their signals through
the transcription factors Stat5a and 5b (referred to as Stat5). However, ... Cited by 11 - Related articles - All 4 versions
- ►endocrinology-journals.org GF Anhe, TCA Nogueira, JE Nicoletti-Carvalho … - Journal of Endocrinology, 2007 - Soc Endocrinology During pregnancy, the maternal endocrine pancreas undergoes, as a consequence of
placental lactogens and prolactin (PRL) action, functional changes that are
characterized by increased glucose-induced insulin secretion. After ... Cited by 10 - Related articles - BL Direct - All 2 versions
- ►endojournals.org PT Fueger, JC Schisler, D Lu, DA Babu, RG … - Molecular Endocrinology, 2008 - Endocrine Soc Both major forms of diabetes involve a decline in β-cell mass, mediated by
autoimmune destruction of insulin-producing cells in type 1 diabetes and by
increased rates of apoptosis secondary to metabolic stress in type 2 ... Cited by 9 - Related articles - BL Direct - All 5 versions
- ►shouxi.net Y Fujinaka, K Takane, H Yamashita, RC … - Journal of Biological Chemistry, 2007 - ASBMB One of the goals in the treatment for diabetes is to enhance pancreatic beta
cell function, proliferation, and survival. This study explores the role of
lactogenic hormones, prolactin (PRL) and placental lactogen (PL), in beta ... Cited by 14 - Related articles - All 4 versions
SC Crepaldi-Alves, EM Carneiro, JR … - Brazilian Journal of Medical and Biological Research, 1997 - SciELO Brasil We studied the synergistic effect of glucose and prolactin (PRL) on insulin
secretion and GLUT2 expression in cultured neonatal rat islets. After 7 days in
culture, basal insulin secretion (2.8 mM glucose) was similar in control ... Cited by 10 - Related articles - Cached - BL Direct - All 9 versions
- ►endocrinology-journals.org J Jensen, ED Galsgaard, AE Karlsen, YC Lee, … - Journal of Endocrinology, 2005 - Soc Endocrinology The proinflammatory cytokines interleukin-1ß (IL-1ß), interferon- (IFN- ) and
tumour necrosis factor- (TNF- ) are toxic to pancreatic ß-cells and are
implicated in the pathogenesis of type 1 diabetes. We have previously found ... Cited by 15 - Related articles - BL Direct - All 3 versions
- ►endojournals.org M Jackerott, YC Lee, K Mollgard, H Kofod, J … - Endocrinology, 2006 - Endocrine Soc Trefoil factors (TFFs) 1, 2, and 3 are expressed in mucosal epithelia. TFFs are
particular abundant in the intestine in which they play a crucial role in
maintenance and restitution of the epithelium. Because pancreas ... Cited by 12 - Related articles - BL Direct - All 4 versions
- ►nih.gov H Mziaut, S Kersting, KP Knoch, WH Fan, M … - Proceedings of the National Academy of Sciences, 2008 - National Acad Sciences Changes in metabolic demands dynamically regulate the total mass of adult
pancreatic β-cells to adjust insulin secretion and preserve glucose
homeostasis. Glucose itself is a major regulator of β-cell proliferation ... Cited by 15 - Related articles - BL Direct - All 7 versions
