- ►diabetesjournals.org I Bady, N Marty, M Dallaporta, M Emery, J Gyger, D … - Diabetes, 2006 - Am Diabetes Assoc A role for glucose in the control of feeding has been proposed, but its precise physiological importance
is unknown. Here, we evaluated feeding behavior in glut2-null mice, which express a transgenic
glucose transporter in their β-cells to rescue insulin secretion (ripglut1;glut2 −/− mice). We ... Cited by 22 - Related articles - BL Direct - All 6 versions
N Marty, I Bady, B Thorens - Diabetes, 2006 - Am Diabetes Assoc The pancreatic β-cell paradigm for glucose sensing has been proposed to apply to brain glucose
sensors controlling counterregulation to hypoglycemia and feeding behavior. Over recent
years, we tested this model in mice by first showing that inactivation of the GLUT2 gene ... Cited by 8 - Related articles - BL Direct - All 4 versions
CV Mobbs, F Isoda, H Makimura, J Mastaitis, T Mizuno, … - Physiology & behavior, 2005 - Elsevier Since nutrition-sensitive feedback signals normally act to maintain relatively stable levels of both
available and stored nutritional resources, failure in one or more of these feedback signals could
plausibly lead to obese phenotypes. The glucostatic hypothesis in its original form posited ... Cited by 22 - Related articles - All 4 versions
- ►nih.gov N Marty, M Dallaporta, M Foretz, M Emery, … - Journal of Clinical …, 2005 - Am Soc Clin Investig Ripglut1;glut2 –/– mice have no endogenous glucose transporter type 2 (glut2) gene expression
but rescue glucose-regulated insulin secretion. Control of glucagon plasma levels is,
however, abnormal, with fed hyperglucagonemia and insensitivity to physiological hypo- ... Cited by 38 - Related articles - BL Direct - All 10 versions
M Arluison, M Quignon, B Thorens, C Leloup, L … - Journal of Chemical …, 2004 - Elsevier Following a former immunohistochemical study in the rat brain [Arluison, M., Quignon, M.,
Nguyen, P., Thorens, B., Leloup, C., Pénicaud, L. Distribution and anatomical localization of the
glucose transporter 2 (GLUT2) in the adult rat brain. I. Immunohistochemical study. J. ... Cited by 19 - Related articles - All 2 versions
M Arluison, M Quignon, P Nguyen, B Thorens, C … - Journal of Chemical …, 2004 - Elsevier The aim of this work was to study the distribution and cellular localization of GLUT2 in the rat
brain by light and electron microscopic immunohistochemistry, whereas our ultrastructural observations
will be reported in a second paper. Confirming previous results, we show that GLUT2- ... Cited by 22 - Related articles - All 2 versions
[CITATION] The Hepatoportal Glucose Sensor-Mechanisms of Glucose Sensing and Signal …
R Burcelin, B Thorens - Diabetes, 2001 - Am Diabetes Assoc GLUT2 −/− mice reexpressing GLUT1 or GLUT2 in their β-cells (RIPGLUT1 × GLUT2 −/− or RIPGLUT2
× GLUT2 −/− mice) have nearly normal glucose-stimulated insulin secretion but show high glucagonemia
in the fed state. Because this suggested impaired control of glucagon secretion, we set ... Cited by 27 - Related articles - BL Direct - All 4 versions
B Li, X Xi, DS Roane, DH Ryan, RJ Martin - Molecular Brain Research, 2003 - Elsevier GK, GLUT2, SUR1, GLP-1R and NPY have been identified in a number of brain areas [3, 4,
7, 9, 11, 12, 14 and 15]. GK has been proposed as a mediator of glucose sensing in the brain
[3 and 19]. GLUT2 has been proposed to play a role in central regulation of food intake ... Cited by 23 - Related articles - All 3 versions
[CITATION] Duong-Van-Huyen JP, Bourahla K, Laugier P, Lelievre-Pegorier M, et al.(2006) …
