KM Boini, AM Hennige, DY Huang, B Friedrich, M … - Diabetes, 2006 - Am Diabetes Assoc Excess salt intake decreases peripheral glucose uptake, thus impairing glucose tolerance. Stimulation
of cellular glucose uptake involves phosphatidylinositide-3-kinase (PI-3K)–dependent activation
of protein kinase B/Akt. A further kinase downstream of PI-3K is serum- and glucocorticoid- ... Cited by 13 - Related articles - BL Direct - All 6 versions
S Jeyaraj, C Boehmer, F Lang, M Palmada - Biochemical and Biophysical …, 2007 - Elsevier Insulin stimulates glucose transport into muscle and fat cells by enhancing GLUT4 abundance
in the plasma membrane through activation of phosphatidylinositol 3-kinase (PI3K). Protein kinase
B (PKB) and PKCζ are known PI3K downstream targets in the regulation of GLUT4. The ... Cited by 10 - Related articles - All 5 versions
- ►diabetesjournals.org M Palmada, C Boehmer, A Akel, J Rajamanickam, S … - Diabetes, 2006 - Am Diabetes Assoc Phosphatidylinositol 3-kinase (PI3 kinase) inhibition disrupts the ability of insulin to stimulate
GLUT1 and GLUT4 translocation into the cell membrane and thus glucose transport. The effect
on GLUT4 but not on GLUT1 is mediated by activation of protein kinase B (PKB). The ... Cited by 24 - Related articles - BL Direct - All 5 versions
F von Wowern, C Fava, G Berglund, O Melander - American Journal of …, 2005 - Elsevier The aim of this study was to investigate if genetic variation in the SGK-1 gene is associated with
increased blood pressure and strength of the insulin-blood pressure correlation. Two SGK1 SNP's
(Genebank accession number “rs1743966” in intron 6 (T/C) and “rs1057293” in exon 8 ... Cited by 29 - Related articles - BL Direct - All 5 versions
- ►physiology.org F Grahammer, F Artunc, D Sandulache, R … - American Journal of …, 2006 - Am Physiological Soc Serum- and glucocorticoid-inducible kinase (SGK) 1 and SGK3 share the ability to upregulate
several ion channels, including the epithelial Na + channel. Whereas SGK1 is under genomic
control of mineralocorticoids and glucocorticoids, SGK3 is constitutively expressed. The ... Cited by 12 - Related articles - BL Direct - All 2 versions
- ►physiology.org [PDF] DY Huang, KM Boini, H Osswald, B … - American Journal of …, 2006 - Am Physiological Soc Page 1. RESISTANCE OF MICE LACKING THE SERUM AND GLUCOCORTICOID
INDUCIBLE KINASE SGK1 AGAINST SALT-SENSITIVE HYPERTENSION INDUCED
BY HIGH FAT DIET Dan Yang Huang 1* , Krishna M. Boini ... Cited by 20 - Related articles - BL Direct - All 6 versions
D Sandulache, F Grahammer, F Artunc, G Henke, A … - Pflügers Archiv European …, 2006 - Springer Page 1. Pflugers Arch - Eur J Physiol (2006) 452: 444–452 DOI 10.1007/s00424-005-0021-x
RENAL FUNCTION, BODY FLUIDS Diana Sandulache . Florian Grahammer . Ferruh Artunc .
Guido Henke . Azeemudeen Hussain . Omaima Nasir . Andreas Mack . Björn Friedrich . ... Cited by 9 - Related articles - BL Direct - All 4 versions
- ►physiology.org F Grahammer, G Henke, C Sandu, R … - American Journal of …, 2006 - Am Physiological Soc In vitro experiments have revealed the ability of serum- and glucocorticoid-inducible kinase 1
(SGK1) to stimulate intestinal Na + -coupled glucose cotransporter 1 (SGLT1) and intestinal Na
+ /H + exchanger 3 (NHE3). The present study explored the contribution of SGK1 to the ... Cited by 22 - Related articles - BL Direct - All 4 versions