- ►diabetesjournals.org AI Tarasov, CAJ Girard, FM Ashcroft - Diabetes, 2006 - Am Diabetes Assoc ATP-sensitive K + channels (K ATP channels) couple cell metabolism to electrical
activity and thereby to physiological processes such as hormone secretion,
muscle contraction, and neuronal activity. However, the mechanism by which ... Cited by 14 - Related articles - BL Direct - All 5 versions
DU Schulze, M Düfer, B Wieringa, P Krippeit … - Diabetologia, 2007 - Springer Abstract Aims/hypothesis In a previous study, we demonstrated that a creatine
kinase (CK) modulates K ATP channel activity in pancreatic beta cells. To
explore phosphotransfer signalling pathways in more detail, we examined ... Cited by 8 - Related articles - BL Direct - All 6 versions
AM Patch, SE Flanagan, C Boustred, AT … - Diabetes Obes Metab, 2007 - interscience.wiley.com It is also possible that your web browser is not configured or not able to
display style sheets. In this case, although the visual presentation will be
degraded, the site should continue to be functional. We recommend using the ... Cited by 9 - Related articles - All 3 versions
P Krippeit-Drews, M Bäcker, M Düfer, G … - Pflügers Archiv European Journal of Physiology, 2003 - Springer Abstract The aim of the present study was to test the hypothesis that a creatine
kinase/phosphocreatine system is involved in the regulation of K ATP channels in
pancreatic b-cells. The phosphocreatine concentration in isolated mouse ... Cited by 17 - Related articles - BL Direct - All 3 versions
GA Rutter - Development of the Pancreas and Neonatal Diabetes: …, 2007 - books.google.com Shield JPH, Scharfmann R (eds): Development of the Pancreas and Neonatal
Diabetes. Endocr Dev. Basel, Karger, 2007, vol 12, pp 75-85 Generating New
Candidate Genes for Neonatal Diabetes: Functional and Genetic Studies of ... Cited by 3 - Related articles - BL Direct - All 4 versions
- ►oxfordjournals.org P Proks, K Shimomura, TJ Craig, CAJ Girard, … - Human Molecular Genetics, 2007 - Oxford Univ Press Activating mutations in the genes encoding the ATP-sensitive potassium (K ATP )
channel subunits Kir6.2 and SUR1 are a common cause of neonatal diabetes. Here,
we analyse the molecular mechanism of action of the heterozygous mutation ... Cited by 12 - Related articles - BL Direct - All 4 versions
- ►shouxi.net R Masia, DD De Leon, C MacMullen, H … - Diabetes, 2007 - Am Diabetes Assoc RESULTS—L225P-containing K ATP channels were significantly more active in the
intact cell than in wild-type channels. In excised membrane patches, L225P
increased channel sensitivity to stimulatory Mg nucleotides without ... Cited by 12 - Related articles - BL Direct - All 6 versions
- ►shouxi.net S Thore, A Wuttke, A Tengholm - Diabetes, 2007 - Am Diabetes Assoc Phosphatidylinositol-4,5-bisphosphate (PIP 2 ) is important for a variety of
cellular processes as a precursor for second messengers and by regulating ion
channels, the cytoskeleton, and vesicle traffic in many types of cells, ... Cited by 5 - Related articles - BL Direct - All 6 versions
- ►nih.gov CW Lin, F Yan, S Shimamura, S Barg, SL … - Diabetes, 2005 - Am Diabetes Assoc ATP-sensitive K + channels (K ATP channels) of pancreatic β-cells play key
roles in glucose-stimulated insulin secretion by linking metabolic signals to
cell excitability. Membrane phosphoinositides, in particular ... Cited by 16 - Related articles - BL Direct - All 6 versions
- ►shouxi.net AI Tarasov, HJ Welters, S Senkel, GU Ryffel, … - Diabetes, 2006 - Am Diabetes Assoc ATP-sensitive K + channels (K ATP channels) couple ß-cell metabolism to
electrical activity and thereby play an essential role in the control of insulin
secretion. Gain-of-function mutations in Kir6.2 (KCNJ11), the pore-forming ... Cited by 17 - Related articles - BL Direct - All 5 versions
