- ►diabetesjournals.org KM Myint, Y Yamamoto, T Doi, I Kato, A … - Diabetes, 2006 - Am Diabetes Assoc Diabetic nephropathy is a major microvascular complication in long-standing
diabetic patients who eventually undergo renal dialysis or transplantation. To
prevent development of this disease and to improve advanced kidney injury, ... Cited by 35 - Related articles - BL Direct - All 6 versions
D Suzuki, M Toyoda, N Yamamoto, M … - Internal Medicine, 2006 - J-STAGE Objective The receptor for advanced glycation end-products (RAGE) is one of
several advanced glycation end-product (AGE)-specific cellular receptors. To
evaluate the relationship between AGE and RAGE in renal tissues of diabetic ... Cited by 21 - Related articles - All 3 versions
R Inagi, Y Yamamoto, M Nangaku, N Usuda, H … - Diabetes, 2006 - Am Diabetes Assoc Many factors are involved in the pathogenesis of diabetic nephropathy. A single
gene abnormality may be prerequisite but insufficient to the disease to
manifest. It is therefore only when a second or sometimes a third damage is ... Cited by 25 - Related articles - BL Direct - All 6 versions
T Shoji, H Koyama, T Morioka, S Tanaka, A … - Diabetes, 2006 - Am Diabetes Assoc Angiogenic response is impaired in diabetes. Here, we examined the involvement
of receptor for advanced glycation end products (RAGE) in diabetes-related
impairment of angiogenesis in vivo. Angiogenesis was determined in ... Cited by 27 - Related articles - BL Direct - All 5 versions
- ►oxfordjournals.org L Gu, S Hagiwara, Q Fan, M Tanimoto, M … - Nephrology Dialysis Transplantation, 2006 - ERA-EDTA Background. Upregulation of local monocyte chemoattractant protein-1 (MCP-1)
production is involved in glomerular damage through macrophage recruitment and
activation in diabetic nephropathy. Treatment of db/db mice with soluble ... Cited by 40 - Related articles - BL Direct - All 5 versions
- ►endocrinology-journals.org LJN Jensen, L Denner, BF Schrijvers, RG … - Journal of Endocrinology, 2006 - Soc Endocrinology Advanced glycation endproducts (AGEs) have been implicated in the pathogenesis
of diabetic kidney disease. The actions of AGEs are mediated both through a
non-receptor mediated pathway and through specific receptors for AGEs (eg ... Cited by 17 - Related articles - BL Direct - All 4 versions
A Harashima, Y Yamamoto, C Cheng, K … - Biochemical Journal, 2006 - pubmedcentral.nih.gov The cell-surface RAGE [receptor for AGE (advanced glycation end-products)] is
associated with the development of diabetic vascular complications,
neurodegenerative disorders and inflammation. Recently, we isolated a human ... Cited by 18 - Related articles - BL Direct - All 7 versions
- ►asnjournals.org J Guo, R Ananthakrishnan, W Qu, Y Lu, N … - Journal of the American Society of Nephrology, 2008 - Am Soc Nephrol In the kidney, the receptor for advanced glycation end products (RAGE) is
principally expressed in the podocyte at low levels, but is upregulated in both
human and mouse glomerular diseases. Because podocyte injury is central to ... Cited by 5 - Related articles - All 5 versions
KCB Tan, SWM Shiu, WS Chow, L Leng, R … - Diabetologia, 2006 - Springer Abstract Aims/hypothesis Activation of the receptor for advanced glycation end
products (RAGE, also known as AGE- specific receptor [AGER]) has been implicated
in the development of diabetic vascular complications. Blockade of RAGE ... Cited by 39 - Related articles - All 5 versions
- ►nih.gov E Harja, D Bu, BI Hudson, JS Chang, X Shen, … - Journal of Clinical Investigation, 2007 - Am Soc Clin Investig Endothelial dysfunction is a key triggering event in atherosclerosis. Following
the entry of lipoproteins into the vessel wall, their rapid modification results
in the generation of advanced glycation endproduct epitopes and subsequent ... Cited by 39 - Related articles - BL Direct - All 5 versions
