- ►diabetesjournals.org J Girard - Diabetes, 2006 - Am Diabetes Assoc Previous studies suggest that insulin can inhibit hepatic glucose production by both direct and
indirect actions. The indirect effects include inhibition of glucagon secretion, reduction in plasma
nonesterified fatty acid levels, reduction of the amount of gluconeogenic precursor ... Cited by 11 - Related articles - BL Direct - All 5 versions
- ►nih.gov [PDF] J Girard - Journal of Clinical Investigation, 2006 - Am Soc Clin Investig Previous studies suggest that insulin can inhibit hepatic glucose production (HGP) by both direct
and indirect actions. The indirect effects include inhibition of glucagon secretion, reduction in
plasma nonesterified fatty acid levels, reduction of the amount of gluconeogenic ... Cited by 9 - Related articles - BL Direct - All 10 versions
J Girard - Journal of Clinical Investigation, 2006 - ukpmc.ac.uk Previous studies suggest that insulin can inhibit hepatic glucose production (HGP) by both direct
and indirect actions. The indirect effects include inhibition of glucagon secretion, reduction in
plasma nonesterified fatty acid levels, reduction of the amount of gluconeogenic ... Related articles
- ►nih.gov AD Cherrington - Journal of Clinical Investigation, 2005 - Am Soc Clin Investig The inability of insulin to suppress hepatic glucose production (HGP) is a key defect found in
type 2 diabetes. Insulin inhibits HGP through both direct and indirect means, the latter of which
include inhibition of glucagon secretion, reduction in plasma nonesterified fatty acid level, ... Cited by 22 - Related articles - All 14 versions
A Roy - 2008 - etd.library.pitt.edu Page 1. DYNAMIC MODELING OF FREE FATTY ACID, GLUCOSE, AND INSULIN DURING REST
AND EXERCISE IN INSULIN DEPENDENT DIABETES MELLITUS PATIENTS by Anirban Roy
BS, University of Pune, India, 2001 MS, University of Arkansas, 2004 ... Related articles - View as HTML - All 4 versions
S Guo, KD Copps, X Dong, S Park, Z Cheng, … - Molecular and Cellular …, 2009 - Am Soc Microbiol We used a Cre-loxP approach to generate mice with varied expression of hepatic Irs1 and Irs2
to establish the contribution of each protein to hepatic nutrient homeostasis. While nutrient-sensitive
transcripts were expressed nearly normally in liver lacking Irs2 (LKO2 mice), these ... Related articles - All 3 versions
YC Lu, E Rozengurt, E Zhukova - Biochemical and Biophysical Research …, 2007 - Elsevier We have previously produced transgenic G-InsKi mice, a model allowing regulated portal insulin
delivery from gastric G cells without using β cells. Here, we report that in G-InsKi mice portal levels
of transgenic human insulin are 6-fold higher than in peripheral circulation. Peptone- ... Related articles - All 5 versions
[CITATION] La régulation du métabolisme du glucose par la protéine tyrosine phosphatase …
