- ►diabetesjournals.org S Kim, HS Kim, KW Chung, SH Oh, JW Yun, … - Diabetes, 2007 - Am Diabetes Assoc RESULTS—STAT1 −/− islet cells were resistant to death by interferon
(IFN)-γ/tumor necrosis factor (TNF)-α or IFN-γ/interleukin (IL)-1β
combination. Cytochrome c translocation by IFN-γ/TNF-α was abrogated in ... Cited by 8 - Related articles - BL Direct - All 6 versions
- ►diabetesjournals.org HI Callewaert, CA Gysemans, L Ladrière, W D … - Diabetes, 2007 - Am Diabetes Assoc OBJECTIVE—Exposure of β-cells to inflammatory cytokines leads to apoptotic
cell death through the activation of gene networks under the control of specific
transcription factors, such as interferon-γ–induced signal transducer ... Cited by 13 - Related articles - BL Direct - All 6 versions
- ►uva.nl [PDF] TC Schreuder, HC Gelderblom, CJ Weegink, … - Liver International, 2008 - medscape.com We screened for DM during and after treatment with pegylated-IFN-α (Peg-IFN-α)
and Riba for chronic HCV infection. This started after we observed two index
cases of DM during Peg-IFN-α/Riba treatment in our hospital in December ... Cited by 8 - Related articles - BL Direct - All 9 versions
- ►jimmunol.org X Wang, S Jia, R Geoffrey, R Alemzadeh, S … - The Journal of Immunology, 2008 - Am Assoc Immnol Understanding active proinflammatory mechanisms at and before type 1 diabetes
mellitus (T1DM) onset is hindered in humans, given that the relevant tissues are
inaccessible and pancreatic immune responses are difficult to measure in ... Cited by 11 - Related articles - All 2 versions
E Lien, D Zipris - Current Molecular Medicine, 2009 - ingentaconnect.com 1 Department of Medicine, University of Massachusetts Medical School, Worcester,
MA 01655, USA 2 Department of Pediatrics, Barbara Davis Center for Childhood
Diabetes, University of Colorado, Aurora, CO 80045, USA Abstract: Toll-like ... Cited by 5 - Related articles - All 2 versions
H Mori, T Shichita, Q Yu, R Yoshida, M … - Biochemical and Biophysical Research …, 2007 - Elsevier Type 1 diabetes results from the selective destruction of insulin-producing
pancreatic β-cells during islet inflammation, which involves inflammatory
cytokines and free radicals. However, mechanisms for protecting β-cells ... Cited by 7 - Related articles - All 5 versions
- ►amjpathol.org AS Mohamood, ML Guler, Z Xiao, D Zheng, A … - American Journal of Pathology, 2007 - ASIP Spontaneous mutation of Fas (lpr) or FasL (gld) completely protects nonobese
diabetic mice from autoimmune diabetes but also causes massive double-negative
T-cell lymphoproliferation. In this study, we used bone marrow chimeras and ... Cited by 6 - Related articles - BL Direct - All 11 versions
- ►jimmunol.org D Vallois, CH Grimm, P Avner, C Boitard, UC … - The Journal of Immunology, 2007 - Am Assoc Immnol The Idd6 locus on mouse chromosome 6, which controls the development of type 1
diabetes in the NOD mouse, affects proliferation rates of T cells and the
activity of regulatory CD4 + CD25 + T cells. Using a transcriptional ... Cited by 7 - Related articles - All 4 versions
- ►jimmunol.org T Raine, P Zaccone, P Mastroeni, A Cooke - The Journal of Immunology, 2006 - Am Assoc Immnol Infection, commencing across a wide age range, with a live, attenuated strain of
Salmonella typhimurium, will halt the development of type 1 diabetes in the NOD
mouse. The protective mechanism appears to involve the regulation of ... Cited by 8 - Related articles - BL Direct - All 6 versions
