- ►diabetesjournals.org YG Chen, PA Silveira, MA Osborne, HD … - Diabetes, 2007 - Am Diabetes Assoc The H2 g7 (K d , A g7 , E null , and D b ) major histocompatibility complex
(MHC) is the primary genetic contributor to type 1 diabetes in NOD mice. NOD
stocks congenically expressing other MHC haplotypes such as H2 nb1 (K b , A ... Cited by 2 - Related articles - BL Direct - All 5 versions
- ►jimmunol.org DV Serreze, MA Osborne, YG Chen, HD … - The Journal of Immunology, 2006 - Am Assoc Immnol In both humans and NOD mice, particular combinations of MHC genes provide the
primary risk factor for development of the autoreactive T cell responses causing
type 1 diabetes (T1D). Conversely, other MHC variants can confer dominant ... Cited by 5 - Related articles - BL Direct - All 7 versions
KA Finucane, CB Archer - Clinical and experimental dermatology, 2006 - 万方数据资源系统 This article is the fourth in a series of CPD articles aimed at reviewing the
past 2 years of the general medical literature, selecting topics that may be of
interest to dermatologists. This issue looks at recent articles on the ... Cited by 3 - Related articles - BL Direct - All 5 versions
P Hořín, J Matiasöovic - Animal Genetics - interscience.wiley.com It is also possible that your web browser is not configured or not able to
display style sheets. In this case, although the visual presentation will be
degraded, the site should continue to be functional. We recommend using the ... Cited by 4 - Related articles - BL Direct - All 2 versions
CB Stober, S Brode, JK White, JF Popoff, JM … - Infection and Immunity, 2007 - Am Soc Microbiol Page 1. 1 Slc11a1 (formerly Nramp1) is expressed in dendritic cells and influences
MHC class II expression and antigen presenting cell function ... Cited by 4 - Related articles
EE HAMILTON-WILLIAMS, X MARTINEZ, M … - ANNALS-NEW YORK ACADEMY OF SCIENCES, 2007 - interscience.wiley.com Abstract : Type 1 diabetes (T1D) occurs because of lack of T cell tolerance to
islet antigens. We hypothesized that critical genetic susceptibility loci that
control progression to T1D, designated as insulin-dependent diabetes (Idd) ... Cited by 4 - Related articles - BL Direct - All 6 versions
G Zekavat, SY Rostami, A Badkerhanian, RF … - The Journal of Immunology, 2008 - Am Assoc Immnol B lymphocytes are required for the pathogenesis of autoimmune diabetes in NOD
mice. Previous studies established that a lymphopenic transitional (TR) B cell
compartment reduces the competitive constraint on the entry of newly ... Cited by 6 - Related articles - All 4 versions
- ►rupress.org AK Panigrahi, NG Goodman, RA Eisenberg, MR … - Journal of Experimental Medicine, 2008 - jem.rupress.org Continued antibody gene rearrangement, termed receptor editing, is an important
mechanism of central B cell tolerance that may be defective in some autoimmune
individuals. We describe a quantitative assay for recombining sequence (RS) ... Cited by 5 - Related articles - All 6 versions
- ►jimmunol.org EA Johnson, P Silveira, HD Chapman, EH … - The Journal of Immunology, 2001 - Am Assoc Immnol Transgenic restoration of normally absent H2-E MHC class II molecules on APC
dominantly inhibits T cell-mediated autoimmune diabetes (IDDM) in nonobese
diabetic (NOD) mice. We analyzed the minimal requirements for transgenic ... Cited by 20 - Related articles - BL Direct - All 2 versions
- ►nih.gov SA Litherland, TX Xie, KM Grebe, A Davoodi- … - Journal of autoimmunity, 2005 - Elsevier Autocrine granulocyte macrophage-colony stimulating factor (GM-CSF) sequentially
activates intracellular components in monocyte/macrophage production of the
pro-inflammatory and immunoregulatory prostanoid, prostaglandin E2 (PGE2). ... Cited by 6 - Related articles - All 4 versions
