- ►diabetesjournals.org B Qin, W Qiu, RK Avramoglu, K Adeli - Diabetes, 2007 - Am Diabetes Assoc There is growing evidence suggesting intestinal insulin resistance and overproduction of apolipoprotein
(apo) B48–containing chylomicrons in insulin-resistant states. In the current study, we investigated
the potential role of the inflammatory cytokine tumor necrosis factor-α (TNF-α) in the ... Cited by 17 - Related articles - BL Direct - All 5 versions
- ►diabetesjournals.org LM Federico, M Naples, D Taylor, K Adeli - Diabetes, 2006 - Am Diabetes Assoc Postprandial dyslipidemia is recognized as an important complication of insulin-resistant
states, and recent evidence implicates intestinal lipoprotein overproduction as a causative
factor. The mechanisms linking intestinal lipoprotein overproduction and aberrant insulin ... Cited by 24 - Related articles - BL Direct - All 5 versions
- ►diabetesjournals.org M Zoltowska, E Ziv, E Delvin, D Sinnett, R Kalman, C … - Diabetes, 2003 - Am Diabetes Assoc Although postprandial hypertriglyceridemia is a major contributing factor in the development
of atherosclerosis, little information is available on the effect of insulin resistance and diabetes
on intestinal fat transport. The aim of the present study was to examine intracellular ... Cited by 24 - Related articles - BL Direct - All 4 versions
J Hsieh, AA Hayashi, J Webb, K Adeli - Atherosclerosis (Supplements)( …, 2008 - Elsevier Insulin resistance is strongly associated with metabolic dyslipidemia, which is largely a postprandial
phenomenon. Though previously regarded as a consequence of delayed triglyceride-rich lipoprotein
clearance, emerging evidence present intestinal overproduction of apoB-48-containing ... Cited by 4 - Related articles - All 25 versions
- ►jlr.org C Zheng, K Ikewaki, BW Walsh, FM Sacks - The Journal of Lipid Research, 2006 - ASBMB We aimed to identify mechanisms by which apolipoprotein B-48 (apoB-48) could have an atherogenic
role by simultaneously studying the metabolism of postprandial apoB-48 and apoB-100
lipoproteins. The kinetics of apoB-48 and apoB-100, each in four density subfractions of ... Cited by 5 - Related articles - BL Direct - All 5 versions
- ►atn.edu.au [PDF] EM Allister, AP James, GF Watts, PHR Barrett, … - European journal of …, 2006 - pt.wkhealth.com Abbreviations: Apo, apolipoprotein; AUC, area under the curve; BMI, body mass index; GIP, gastric
inhibitory peptide; HDL, high density lipoprotein; HOMA, homeostatic model assessment;
IAUC, incremental area under the curve; IMGD, insulin mediated glucose disposal; LDL, ... Cited by 2 - Related articles - BL Direct - All 6 versions
B Qin, M Nagasaki, M Ren, G Bajotto, Y Oshida, Y Sato - Life Sciences, 2003 - Elsevier This study investigated the effects of the traditional herbal medicine, Keishi-ka-jutsubu-to
(KJT) on insulin action in vivo and insulin signaling in skeletal muscle in STZ-induced
diabetes. Rats were divided into single and 7-days oral administration groups. ... Cited by 14 - Related articles - All 5 versions
JD Sparks, CE Sparks - The Journal of Clinical Investigation, 2008 - pubmedcentral.nih.gov Excessive production of triglyceride-rich VLDL, which can result from dietary
overindulgence, underlies metabolic syndrome — a combination of disorders including high
blood pressure, obesity, high triglyceride, and insulin resistance — and places individuals ... Cited by 5 - Related articles - BL Direct - All 7 versions
- ►endojournals.org GF Lewis, M Naples, K Uffelman, N Leung, L Szeto, K … - Endocrinology, 2004 - Endocrine Soc It is not known whether intestinal lipoprotein production is stimulated by an acute elevation of
plasma free fatty acids (FFA). We examined the effect of an intralipid and heparin infusion on
the intestinal lipoprotein production rate (PR) in insulin-sensitive [chow-fed (CHOW)], ... Cited by 18 - Related articles - BL Direct - All 4 versions
- ►physiology.org B Qin, RA Anderson, K Adeli - American Journal of Physiology- …, 2008 - Am Physiological Soc Insulin-resistant states are commonly associated with both increased circulating levels of tumor
necrosis factor (TNF)- and hepatic overproduction of very low density lipoproteins (VLDL).
Here, we provide evidence that increased TNF- can directly stimulate the hepatic ... Cited by 6 - Related articles - BL Direct - All 5 versions
