- ►shouxi.net UA White, AA Coulter, TK Miles, JM Stephens - Diabetes, 2007 - Am Diabetes Assoc The purpose of this study was to determine whether pyruvate dehydrogenase kinase (PDK)4 was
expressed in adipocytes and whether PDK4 expression was hormonally regulated in fat cells. Both
Northern blot and Western blot ... Cited by 13 - Related articles - BL Direct - All 5 versions
JC Hogan, JM Stephens - Diabetes, 2005 - Am Diabetes Assoc Growth hormone (GH) diminishes adipose tissue mass in vivo and decreases expression and activity of
fatty acid synthase (FAS) in adipocytes. GH and prolactin (PRL) are potent activators of STAT5 and
exert adipogenic and antiadipogenic ... Cited by 31 - Related articles - All 5 versions
UA White, AA Coulter, TK Miles, JM Stephens - Am Diabetes Assoc Page 1. The STAT5A mediated induction of PDK4 expression by prolactin or growth hormone in adipocytes
Received for publication 12 September 2006 and accepted in revised form 19 February 2007. Ursula
A. White, Ann A. Coulter, Tiffany K. Miles and Jacqueline M. Stephens* ... Related articles
[CITATION] Prolactin inhibits expression of B-cell lymphoma 6 (BCL6) in breast cancer through …
TH Tran, FE Utama, J Lin, LM Neilson, AB Sjolund, H … - … of the 90th Annual Meeting of …, 2008 Cited by 3 - Related articles
W Ci, JM Polo, A Melnick - Current opinion in hematology, 2008 - pubmedcentral.nih.gov The BCL6 transcriptional repressor is the most commonly involved oncogene in B-cell lymphomas.
Sustained expression of BCL6 causes malignant transformation of germinal center (GC) B-cells.
Understanding the mechanism of ... Cited by 3 - Related articles - All 3 versions
- ►nih.gov LM Mendez, JM Polo, JJ Yu, M Krupski, BB … - Molecular and Cellular …, 2008 - Am Soc Microbiol The transcription repressor BCL6 plays an essential role in the formation and function of germinal
centers (GCs). While normal B cells promptly shut off BCL6 when they exit the GC, many GC-derived
B-cell lymphomas sustain BCL6 ... Cited by 6 - Related articles - BL Direct - All 10 versions
- ►endocrinology-journals.org M Kawai, N Namba, S Mushiake, Y Etani, R … - Journal of Molecular …, 2007 - Soc Endocrinology Growth hormone-deficient (GHD) patients show a decreased number of adipocytes, which is normalized
by GH replacement, indicating an adipogenic effect of GH. However, the precise mechanisms underlying
this effect remain to be clarified. ... Cited by 6 - Related articles - All 4 versions
- ►endocrinology-journals.org TX Cui, R Kwok, J Schwartz - Journal of Endocrinology, 2008 - Soc Endocrinology GH activates the c-fos promoter by regulating multiple transcription factors. This study adds to our
understanding of GH-regulated transcription by demonstrating that GH regulates the c-fos
cAMP-response element (CRE) and its ... Cited by 8 - Related articles - BL Direct - All 2 versions
- ►endojournals.org V Wauthier, DJ Waxman - Molecular Endocrinology, 2008 - Endocrine Soc Pituitary GH-secretory profiles are sex dependent and regulate the sexually dimorphic expression of
a large number of genes in the liver. The slow response of many sex-specific liver genes to changes
in plasma GH status suggests that ... Cited by 8 - Related articles - All 4 versions
- ►nih.gov TD Brandebourg, JL Bown, N Ben-Jonathan - Biochemical and biophysical …, 2007 - Elsevier Prolactin (PRL) is recognized as a metabolic regulator during lactation, but little information
exists on its actions in male adipose tissue. We examined whether PRL affects the expression of its
receptors (PRLR), lipolysis, and ... Cited by 8 - Related articles - All 7 versions
