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Scholar Results 1 - 10 of about 101 related to del Rincon: Growth Hormone Regulation of p85α Expression and Phosphoinositide 3-Kinase Activity.... (0.09 sec) 

Growth Hormone Regulation of p85α Expression and Phosphoinositide 3-Kinase Activity in …

- diabetesjournals.org
JP del Rincon, K Iida, BD Gaylinn, CE … - Diabetes, 2007 - Am Diabetes Assoc
Phosphoinositide (PI) 3-kinase is involved in insulin-mediated effects on
glucose uptake, lipid deposition, and adiponectin secretion from adipocytes.
Genetic disruption of the p85α regulatory subunit of PI 3-kinase increases ...
Cited by 26 - Related articles - BL Direct - All 7 versions

[PDF] Mechanism for Growth Hormone–Mediated Insulin Resistance


JP del Rincon, K Iida, BD Gaylinn, CE … - DIABETES, 2007 - Am Diabetes Assoc
Page 1. Original Article Growth Hormone Regulation of p85 Expression and
Phosphoinositide 3-Kinase Activity in Adipose Tissue Mechanism ...
Related articles - All 2 versions

Growth Hormone Regulation of p85a Expression and Phosphoinositide 3-Kinase Activity in …


JP del Rincon, K Iida, BD Gaylinn, CE … - Am Diabetes Assoc
Juan-Pablo del Rincon1, Keiji Iida1, Bruce D. Gaylinn1, Carrie E. McCurdy2, J.
Wayne Leitner3, Linda A. Barbour4,5, John J. Kopchick6, Jacob E. Friedman2,
Boris Draznin3,4, and Michael O. ... Thorner1 From the 1Department of ...
Related articles

Increased P85 {alpha} is a potent negative regulator of skeletal muscle insulin signaling …

- jbc.org
LA Barbour, M Rahman… - Journal of Biological Chemistry, 2005 - ASBMB
Insulin resistance is a cardinal feature of normal pregnancy and excess growth
hormone (GH) states, but its underlying mechanism remains enigmatic. We
previously found a significant increase in the p85 regulatory subunit of ...
Cited by 43 - Related articles - BL Direct - All 5 versions

Growth hormone signaling in vivo in human muscle and adipose tissue: impact of insulin, …

- endojournals.org
C Nielsen, LC Gormsen, N Jessen, SB … - Journal of Clinical Endocrinology & Metabolism, 2008 - Endocrine Soc
Intervention: GH was administered as a bolus (n = 8) and constant infusion (n =
8). The bolus study included three arms: 1) control (saline), 2) GH (0.5 mg iv),
and 3) GH blockade (pegvisomant 30 mg sc), each combined with a ...
Cited by 13 - Related articles - All 4 versions

The STAT5A-mediated induction of pyruvate dehydrogenase kinase 4 expression by …

- shouxi.net
UA White, AA Coulter, TK Miles, JM Stephens - Diabetes, 2007 - Am Diabetes Assoc
The purpose of this study was to determine whether pyruvate dehydrogenase kinase
(PDK)4 was expressed in adipocytes and whether PDK4 expression was hormonally
regulated in fat cells. Both Northern blot and Western blot analyses were ...
Cited by 13 - Related articles - BL Direct - All 5 versions

B-cell lymphoma 6 and the molecular pathogenesis of diffuse large B-cell lymphoma


W Ci, JM Polo, A Melnick - Current opinion in hematology, 2008 - pubmedcentral.nih.gov
The BCL6 transcriptional repressor is the most commonly involved oncogene in
B-cell lymphomas. Sustained expression of BCL6 causes malignant transformation
of germinal center (GC) B-cells. Understanding the mechanism of action of ...
Cited by 3 - Related articles - All 3 versions

[CITATION] Prolactin inhibits expression of B-cell lymphoma 6 (BCL6) in breast cancer through a …


TH Tran, FE Utama, J Lin, LM Neilson, AB … - Program of the 90th Annual Meeting of The Endocrine …, 2008
Cited by 3 - Related articles

CtBP is an essential corepressor for BCL6 autoregulation

- nih.gov
LM Mendez, JM Polo, JJ Yu, M Krupski, BB … - Molecular and Cellular Biology, 2008 - Am Soc Microbiol
The transcription repressor BCL6 plays an essential role in the formation and
function of germinal centers (GCs). While normal B cells promptly shut off BCL6
when they exit the GC, many GC-derived B-cell lymphomas sustain BCL6 ...
Cited by 6 - Related articles - BL Direct - All 10 versions

Growth hormone stimulates adipogenesis of 3T3-L1 cells through activation of the Stat5A …

- endocrinology-journals.org
M Kawai, N Namba, S Mushiake, Y Etani, R … - Journal of Molecular Endocrinology, 2007 - Soc Endocrinology
Growth hormone-deficient (GHD) patients show a decreased number of adipocytes,
which is normalized by GH replacement, indicating an adipogenic effect of GH.
However, the precise mechanisms underlying this effect remain to be ...
Cited by 6 - Related articles - All 4 versions


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