- ►diabetesjournals.org N Liadis, L Salmena, E Kwan, P Tajmir, SA Schroer, A … - Diabetes, 2007 - Am Diabetes Assoc Inadequate pancreatic β-cell mass resulting from excessive β-cell apoptosis is a key defect in
type 1 and type 2 diabetes. Caspases are the major molecules involved in apoptosis;
however, in vivo roles of specific caspases in diabetes are unclear. The purpose of this ... Cited by 6 - Related articles - BL Direct - All 4 versions
M Woo - Am Diabetes Assoc Authors: Nicole Liadis (MSc)1, Leonardo Salmena (PhD)1,2, Edwin Kwan (MSc)3, Panteha Tajmir
(MSc)1, Stephanie A. Schroer1, Anna Radziszewska (BSc)1, Xie Li (PhD)3, Laura Sheu
(BSc)3, Mohamed Eweida (PhD)1, Shilong Xu (BSc)1, Herbert Y. Gaisano (MD)3, ... Related articles
… G Dharmadhikari, DM Schumann, J Størling - Expert Opin. Biol. …, 2009 - informahealthcare.com Since having been cloned in 1984, IL-1β has been the subject of over 22,000 citations in
Pubmed, among them over 800 reviews. This is because of its numerous effects. IL-1β is a regulator
of the body's inflammatory response and is produced after infection, injury, and antigenic ... Related articles - All 6 versions
NN Danial - Oncogene, 2008 - nature.com The BH3-only pro-apoptotic proteins are upstream sensors of cellular damage that selectively
respond to specific, proximal death and survival signals. Genetic models and biochemical studies
indicate that these molecules are latent killers until activated through transcriptional or ... Related articles - All 3 versions
P Gao, Y Jiao, Q Xiong, CY Wang, I Gerling, … - Current …, 2008 - pubmedcentral.nih.gov A systematic study has been conducted of all available reports in PubMed and OMIM (Online
Mendelian Inheritance in Man) to examine the genetic and molecular basis of quantitative genetic
loci (QTL) of diabetes with the main focus on genes and polymorphisms. The major ... Related articles - All 4 versions
- ►diabetesjournals.org [PDF] S Klinger, C Poussin, MB Debril, W Dolci, PA Halban, B … - Diabetes, 2007 - Am Diabetes Assoc 1 Institute of Physiology, 2 Center for Integrative Genomics, University of Lausanne, 1015
Lausanne, Switzerland, 3 Department of Genetic Medicine and Development, University Medical
Center, University Hospital, 1211 Geneva 4, Switzerland ... Running title: Increasing ... Cited by 1 - Related articles - All 2 versions
- ►tokushima-u.ac.jp [PDF] K Miyawaki, H Inoue, P Keshavarz, K Mizuta, A … - Diabetes research and …, 2008 - Elsevier In an attempt to rectify the hyperglycemic state in obese insulin resistant db/db mice, a transgenic
line was generated (db/db-CDK4 R24C ) that expresses a constitutively active form of
cyclin-dependent kinase 4 (CDK4/R24C) under the control of the insulin promoter. ... Cited by 1 - Related articles - All 11 versions
- ►nih.gov R Glas, NS Sauter, FT Schulthess, L Shu, J Oberholzer, … - Diabetologia, 2009 - Springer Abstract Aims/hypothesis In obesity, beta cells activate compensatory mechanisms to adapt to
the higher insulin demand. Interleukin-1 receptor antagonist (IL-1Ra) prevents obesity- induced
hyperglycaemia and is a potent target for the treatment of diabetes, but the mechanisms ... Cited by 1 - Related articles - All 5 versions
L Rachdi, N Balcazar, F Osorio-Duque, L Elghazi, A … - PNAS, 2008 - National Acad Sciences *Division of Endocrinology, Metabolism, and Lipid Research, Washington University School
of Medicine, 660 South Euclid Avenue, Campus Box 8127, St. Louis, MO 63110; and ‡ Division
of Medical Genetics, Department of Pediatrics, University of Texas Health Science Center, ... Cited by 1 - Related articles - All 2 versions
LMS Pickersgill, TR Mandrup-Poulsen, UB … - Diabetes/Metabolism … - interscience.wiley.com It is also possible that your web browser is not configured or not able to display style sheets.
In this case, although the visual presentation will be degraded, the site should continue to be
functional. We recommend using the latest version of Microsoft or Mozilla web browser to ... Cited by 1 - Related articles - All 2 versions
