- ►diabetesjournals.org CM Filippi, AE Juedes, JE Oldham, E Ling, L … - Diabetes, 2008 - Am Diabetes Assoc RESULTS—Surprisingly, when expressed selectively in the pancreas, TGF-β
reduced apoptosis of differentiated autoreactive CD8 + T-cells, favoring their
expansion and infiltration of the islets. These results pointed to ... Cited by 4 - Related articles - All 4 versions
- ►diabetesjournals.org [PDF] CM Filippi, AE Juedes, JE Oldham, E Ling, L … - Diabetes, 2008 - Am Diabetes Assoc Christophe M. Filippi,#1 Amy E. Juedes,#1 Janine E. Oldham,1 Ellie Ling,1 Lisa
Togher,1 Yufeng Peng,2 Richard A. Flavell2,3 & Matthias G. von Herrath1 ...
From 1La Jolla Institute for Allergy and Immunology, La Jolla, CA 92037, ... Cited by 1 - Related articles - All 2 versions
- ►jimmunol.org P Truong, DB McGavern - The Journal of Immunology, 2008 - Am Assoc Immnol Restrictions in the diversity of an adaptive immune repertoire can facilitate
viral persistence. Because a host afflicted with an immune deficiency is not
likely to purge a persistent infection using endogenous mechanisms, it is ... Cited by 1 - Related articles - All 4 versions
CM Filippi, EA Estes, JE Oldham, MG von … - The Journal of Clinical Investigation, 2009 - pubmedcentral.nih.gov Type 1 diabetes (T1D) is an autoimmune disease that is caused by the destruction
of insulin-producing β cells. Viral infections induce immune responses that can
damage β cells and promote T1D or on the other hand prevent the ... Cited by 1 - Related articles - All 7 versions
MJ Richer, N Straka, D Fang, I Shanina, MS … - Diabetes, 2008 - Am Diabetes Assoc OBJECTIVE—Coxsackievirus infections have long been associated with the
induction of type 1 diabetes. Infection with coxsackievirus B4 (CB4) enhances
type 1 diabetes onset in NOD mice by accelerating the presentation of ... Cited by 9 - Related articles - BL Direct - All 4 versions
MM Martinic, AE Juedes, D Bresson, D Homann, … - Diabetes, 2007 - Am Diabetes Assoc During an autoimmune process, the autoaggressive response spreads from the
initiating autoantigen to other antigens expressed in the target organ. Based on
evidence from experimental models for multiple sclerosis, such “antigenic ... Cited by 3 - Related articles - BL Direct - All 7 versions
LA Zenewicz, RA Flavell, AL Foundation - European Journal of Immunology, 2008 - interscience.wiley.com IL-22 is a Th17 T-cell-associated cytokine that is highly expressed during
chronic inflammation. IL-22 receptor expression is absent on immune cells, but
is instead restricted to the tissues, providing signaling directionality ... Cited by 3 - Related articles - All 3 versions
[CITATION] CD41FoxP31 regulatory T cells confer infectious tolerance in a TGF-beta-dependent …
- ►pnas.org Y Laouar, T Town, D Jeng, E Tran, Y Wan, VK … - Proceedings of the National Academy of Sciences, 2008 - National Acad Sciences One unresolved issue in immune tolerance is what prevents self-reactive T cells
from activation. In this study, we used a transgenic mouse model of targeted
functional inactivation of TGF-βR signaling in CD11c + cells (CD11c dnR ... Cited by 5 - Related articles - All 5 versions
C Schramm, S Huber, M Protschka, P Czochra, … - International Immunology - Jpn Soc Immunol Page 1. TGFb regulates the CD41CD251 T-cell pool and the expression of Foxp3
in vivo Christoph Schramm 1 , Samuel Huber 1 , Martina ... Cited by 4 - Related articles
