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Scholar Results 1 - 10 of about 101 related to Riboulet-Chavey: Inhibition of AMP-Activated Protein Kinase Protects Pancreatic β-Cells.... (0.09 sec) 

Inhibition of AMP-Activated Protein Kinase Protects Pancreatic β-Cells From Cytokine- …


A Riboulet-Chavey, F Diraison, LK Siew, FS … - Diabetes, 2008 - Am Diabetes Assoc
RESEARCH DESIGN AND METHODS— Clonal MIN6 β-cells or CD1 mouse pancreatic
islets were infected with recombinant adenoviruses encoding enhanced green
fluorescent protein (eGFP/null), constitutively active AMPK (AMPK-CA), or ...
Cited by 12 - Related articles - BL Direct - All 4 versions

[CITATION] Di Leo., V., and Lu, J.(2007)


DM McKay, JL Watson, A Wang, J Caldwell, D … - J. Pharmacol. Exp. Ther
Cited by 2 - Related articles

Transcriptional mechanism of suppression of insulin gene expression by AMP-activated …


JW Kim, JH Cho, SH Ko, H Park, J Ha, KH … - Biochemical and biophysical research communications, 2007 - Elsevier
It is well known that the activation of AMP-activated protein kinase (AMPK)
represses insulin gene expression and glucose-stimulated insulin secretion.
However, how this effect is achieved and the effects of AMPK activation on ...
Cited by 3 - Related articles - All 5 versions

Mechanism of Interferon-γ-Induced Increase in T84 Intestinal Epithelial Tight Junction


MA Boivin, PK Roy, A Bradley, JC Kennedy, T … - Journal of Interferon and Cytokine Research, 2009 - liebertonline.com
Page 1. 45 JOURNAL OF INTERFERON & CYTOKINE RESEARCH Volume 29, Number 1,
2009 © Mary Ann Liebert, Inc. DOI: 10.1089/jir.2008.0128 ...
Cited by 3 - Related articles - All 3 versions

CD8+ T-cells and their interaction with other cells in damage to islet beta-cells


F Wong, L Siew, L Wen - Biochemical Society Transactions, 2008 - biochemsoctrans.org
The autoimmune attack on pancreatic β-cells is orchestrated by a variety of
cells that produce cytokines and other toxic mediators. CD8 + T-cells work
together with other lymphocytes and antigen-presenting cells to mediate ...
Cited by 4 - Related articles - Cached - BL Direct - All 4 versions

Compound C inhibits clonal expansion of preadipocytes by increasing p21 level …


M Nam, WH Lee, EJ Bae, SG Kim - Archives of biochemistry and biophysics, 2008 - Elsevier
AMP-activated protein kinase (AMPK) activation inhibits adipocyte
differentiation. This study investigated the effect of compound C (CC), a widely
used AMPK inhibitor, on differentiation of 3T3-L1 preadipocytes. CC ...
Cited by 5 - Related articles - All 3 versions

The involvement of AMP-activated protein kinases in the anti-inflammatory effect of nicotine in …


PY Cheng, YM Lee, KK Law, CW Lin, MH Yen - Biochemical pharmacology, 2007 - Elsevier
AMP-activated protein kinase (AMPK) is the downstream component of a kinase
cascade that plays a pivotal role in energy homeostasis. AMPK has recently
emerged as an attractive and novel target for inflammatory disorders. Thus, ...
Cited by 8 - Related articles - All 5 versions

Use of a systems biology approach to understand pancreatic beta-cell death in Type 1 …


D Eizirik, F Moore, D Flamez, F Ortis - Biochemical Society Transactions, 2008 - biochemsoctrans.org
Accumulating evidence indicates that β-cells die by apoptosis in T1DM (Type 1
diabetes mellitus). Apoptosis is an active gene-directed process, and recent
observations suggest that β-cell apoptosis depends on the parallel and/or ...
Cited by 9 - Related articles - Cached - BL Direct - All 6 versions

Forskolin-induced cell shrinkage and apical translocation of functional enhanced green …


AM WOOLLHEAD, DL BAINES - The Journal of biological chemistry(Print), 2006 - cat.inist.fr
Elevation of intracellular cAMP increases fluid re-absorption in the lung by
raising amiloride-sensitive Na+  transport through the apically localized
epithelial, amiloride-sensitive Na+  channel(ENaC). However, the ...
Cited by 8 - Related articles - BL Direct - All 2 versions

AICAR potentiates ROS production induced by chronic high glucose: Roles of AMPK in …


WH Kim, JW Lee, YH Suh, HJ Lee, SH Lee, … - Cellular signalling, 2007 - Elsevier
We previously demonstrated that chronic high glucose (33.3 mM) induced β-cell
dysfunction and apoptosis through glucokinase (GCK) downregulation, but the
exact mechanisms involved remain unclear. Here, we show that prolonged ...
Cited by 21 - Related articles - All 5 versions


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