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Scholar Results 1 - 10 of about 101 related to Bucciarelli: RAGE and modulation of ischemic injury in the diabetic myocardium. (0.11 sec) 

RAGE and modulation of ischemic injury in the diabetic myocardium

- nih.gov
… , R Ananthakrishnan, YC Hwang, M Kaneko, F … - Diabetes, 2008 - Am Diabetes Assoc
RESULTS—Diabetic RAGE-null mice were significantly protected from the adverse
impact of I/R injury in the heart, as indicated by decreased release of LDH and
lower glycoxidation products carboxymethyl-lysine (CML) and pentosidine, ...
Cited by 11 - Related articles - All 4 versions

RAGE modulates myocardial injury consequent to LAD infarction via impact on JNK and …

- physiology.org
A Aleshin, R Ananthakrishnan, Q Li, R … - American Journal of Physiology- Heart and Circulatory …, 2008 - Am Physiological Soc
The receptor for advanced glycation end-products (RAGE) has been implicated in
the pathogenesis of ischemia-reperfusion (I/R) injury in the isolated perfused
heart. To test the hypothesis that RAGE-dependent mechanisms modulated ...
Cited by 14 - Related articles - BL Direct - All 3 versions

Oxygen deprivation triggers upregulation of early growth response-1 by the receptor for …

- ahajournals.org
JS Chang, T Wendt, W Qu, L Kong, YS Zou, … - Circulation Research, 2008 - Am Heart Assoc
Myocardial infarction, stroke, and venous thromboembolism are characterized by
oxygen deprivation. In hypoxia, biological responses are activated that evoke
tissue damage. Rapid activation of early growth response-1 in hypoxia ...
Cited by 14 - Related articles - All 5 versions

RAGE ligation affects T cell activation and controls T cell differentiation

- jimmunol.org
Y Chen, EM Akirav, W Chen, O Henegariu, B … - The Journal of Immunology, 2008 - Am Assoc Immnol
The pattern recognition receptor, RAGE, has been shown to be involved in
adaptive immune responses but its role on the components of these responses is
not well understood. We have studied the effects of a small molecule ...
Cited by 10 - Related articles - All 3 versions

Telmisartan Inhibits Advanced Glycation End Products (AGEs)-Elicited Endothelial Cell …


S Yamagishi, T Matsui, K Nakamura, M … - Protein and peptide letters, 2008 - ingentaconnect.com
Abstract: Advanced glycation end products (AGEs)-their receptor (RAGE) axis
plays a central role in the pathogenesis of diabetic microangiopathy. Since the
pathophysiological crosstalk between the AGEs-RAGE system and angiotensin ...
Cited by 5 - Related articles - All 3 versions

Receptor for advanced glycation end products expression on T cells contributes to antigen- …

- jimmunol.org
B Moser, DD Desai, MP Downie, Y Chen, SF … - The Journal of Immunology, 2007 - Am Assoc Immnol
Receptor for advanced glycation end products (RAGE) is an activation receptor
triggered by inflammatory S100/calgranulins and high mobility group box-1
ligands. We have investigated the importance of RAGE on Ag priming of T ...
Cited by 21 - Related articles - All 3 versions

Receptor for advanced glycation end-products (RAGE) modulates neutrophil adhesion and …


F Toure, JM Zahm, R Garnotel, E Lambert, N … - Biochem. J, 2008 - 万方数据资源系统
AGEs (advanced glycation end-products) accumulate in collagen molecules during
uraemia and diabetes, two diseases associated with high susceptibility to
bacterial infection. Because neutrophils bind to collagen during their ...
Cited by 6 - Related articles - All 4 versions

[PDF] Receptor for advanced glycation end products (RAGE) mediates neuronal differentiation and …


L Wang, S Li, FB Jungalwala - Journal of Neuroscience Research, 2008 - tju.edu
The receptor for advanced glycation end products (RAGE) plays a crucial role in
several disease proc- esses, such as diabetes, inflammation, and neurode-
generation. In this article we report multiple roles of RAGE in neuronal ...
Cited by 6 - Related articles - View as HTML - BL Direct - All 5 versions

Methylglyoxal and methylglyoxal-arginine adducts do not directly inhibit endothelial nitric …


O Brouwers, T Teerlink, J van Bezu, R Barto, … - ANNALS-NEW YORK ACADEMY OF SCIENCES, 2008 - interscience.wiley.com
Increased formation of the reactive dicarbonyl compound methylglyoxal (MGO) and
MGO-derived advanced glycation end products (AGEs) seems to be implicated in
endothelial dysfunction and the development of diabetic vascular ...
Cited by 2 - Related articles - BL Direct - All 5 versions

Increased glycation of hemoglobin in chronic renal failure:[corrected] potential role of …


N Selvaraj, Z Bobby, MG Sridhar - Archives of medical research, 2008 - ncbi.nlm.nih.gov
Among the various mechanisms proposed, the process of non-enzymatic glycation of
proteins is believed to play an important role in the pathogenesis of chronic
complications associated with renal failure. The two traditional factors ...
Cited by 2 - Related articles


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