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Scholar Results 1 - 10 of about 101 related to Soro-Paavonen: Receptor for advanced glycation end products (RAGE) deficiency attenuates.... (0.09 sec) 

Receptor for advanced glycation end products (RAGE) deficiency attenuates the development …


A Soro-Paavonen, A Watson, J Li, K Paavonen … - Diabetes, 2008 - Am Diabetes Assoc
RESULTS—Although diabetic apoE −/− mice showed increased plaque
accumulation (14.9 ± 1.7%), diabetic RAGE −/− /apoE −/− mice had
significantly reduced atherosclerotic plaque area (4.9 ± 0.4%) to levels ...
Cited by 22 - Related articles - All 9 versions

RAGE, carboxylated glycans and S100A8/A9 play essential roles in colitis-associated …

- oxfordjournals.org
O Turovskaya, D Foell, P Sinha, T Vogl, R … - Carcinogenesis, 2008 - Oxford Univ Press
Patients with inflammatory bowel diseases are at increased risk for colorectal
cancer, but the molecular mechanisms linking inflammation and cancer are not
well defined. We earlier showed that carboxylated N-glycans expressed on ...
Cited by 18 - Related articles - All 7 versions

Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE …

- nih.gov
E Harja, D Bu, BI Hudson, JS Chang, X Shen, … - Journal of Clinical Investigation, 2007 - Am Soc Clin Investig
Endothelial dysfunction is a key triggering event in atherosclerosis. Following
the entry of lipoproteins into the vessel wall, their rapid modification results
in the generation of advanced glycation endproduct epitopes and subsequent ...
Cited by 39 - Related articles - BL Direct - All 5 versions

High-mobility group box-1 in ischemia-reperfusion injury of the heart

- ahajournals.org
M Andrassy, HC Volz, JC Igwe, B Funke, SN … - Circulation, 2008 - Am Heart Assoc
Methods and Results— Here, we demonstrate that HMGB1 acts as an early mediator
of inflammation and organ damage in I/R injury of the heart. HMGB1 levels were
already elevated 30 minutes after hypoxia in vitro and in ischemic injury ...
Cited by 32 - Related articles - All 7 versions

Posttranslationally modified proteins as mediators of sustained intestinal inflammation

- amjpathol.org
M Andrassy, J Igwe, F Autschbach, C Volz, A … - American Journal of Pathology, 2006 - ASIP
Oxidative and carbonyl stress leads to generation of N
-carboxymethyllysine-modified proteins (CML-mps), which are known to bind the
receptor for advanced glycation end products (RAGE) and induce nuclear ...
Cited by 24 - Related articles - BL Direct - All 7 versions

The HMGB1 receptor RAGE mediates ischemic brain damage

- jneurosci.org
S Muhammad, W Barakat, S Stoyanov, S … - Journal of Neuroscience, 2008 - Soc Neuroscience
In ischemic stroke, the necrotic core is surrounded by a zone of inflammation,
in which delayed cell death aggravates the initial insult. Here, we provide
evidence that the receptor for advanced glycation end products (RAGE) ...
Cited by 24 - Related articles - All 4 versions

Methylglyoxal, diabetes mellitus and diabetic complications


DL Vander Jagt - Drug Metabol Drug Interact, 2008 - 万方数据资源系统
A large literature has developed around methylglyoxal (MG) concerning its role
in diabetes mellitus (DM) and in the development of diabetic complications. This
is related to the observation that levels of reactive aldehydes, especially ...
Cited by 3 - Related articles - BL Direct - All 2 versions

Tempering the wrath of RAGE: An emerging therapeutic strategy against diabetic …


SF Yan, SD Yan, R Ramasamy, AM Schmidt - Annals of Medicine, 2009 - informahealthcare.com
The multiligand receptor RAGE (receptor for advanced glycation end-products) is
emerging as a central mediator in the immune/inflammatory response.
Epidemiological evidence accruing in the human suggests upregulation of ...
Cited by 3 - Related articles - All 3 versions

Maturing dendritic cells depend on RAGE for in vivo homing to lymph nodes

- jimmunol.org
AA Manfredi, A Capobianco, A Esposito, F De … - The Journal of Immunology, 2008 - Am Assoc Immnol
The mobilization of dendritic cells (DCs) from peripheral tissues is critical
for the establishment of T cell-dependent immune responses or tolerance, because
the physical interaction of DCs with naive T cells takes place in the T ...
Cited by 15 - Related articles - All 3 versions

Endothelial dysfunction and vascular inflammation in Type 2 diabetes: interaction of AGE/ …

- physiology.org
A Csiszar, Z Ungvari - American Journal of Physiology- Heart and Circulatory …, 2008 - Am Physiological Soc
In their article, Gao et al. (13) tested the hypothesis that impaired nitric
oxide-dependent dilation in small coronary arteries in Type 2 diabetes results,
in part, from elevated production of superoxide induced by the interaction ...
Cited by 5 - Related articles - All 4 versions


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