- ►diabetesjournals.org K Wanic, G Placha, J Dunn, A Smiles, JH … - Diabetes, 2008 - Am Diabetes Assoc OBJECTIVES— Recently, an association was found between diabetic nephropathy
and the D18S880 microsatellite, located in the carnosinase gene (CNDP1) on
chromosome 18q. Alleles of this microsatellite encode for a variable number ... Cited by 3 - Related articles - All 3 versions
CW McDonough, PJ Hicks, L Lu, CD … - Human Genetics, 2009 - Springer Abstract Four genome wide linkage scans for diabetic nephropathy have mapped
susceptibility loci to chromo- some 18q22.3-23 in the region of the carnosinase
genes, CNDP1 and CNDP2. CNDP1 has been associated with dia- betic ... Cited by 4 - Related articles
- ►shouxi.net E Riedl, H Koeppel, P Brinkkoetter, P Sternik … - Diabetes, 2007 - Am Diabetes Assoc Recently, we demonstrated that a polymorphism in exon 2 of the serum carnosinase
(CNDP1) gene is associated with susceptibility to developing diabetic
nephropathy. Based on the number of CTG repeats in the signal peptide, five ... Cited by 7 - Related articles - BL Direct - All 4 versions
K Wanic, G Placha, J Dunn, A Smiles, JH … - Diabetes, 2008 - pubmedcentral.nih.gov OBJECTIVES— Recently, an association was found between diabetic nephropathy
and the D18S880 microsatellite, located in the carnosinase gene (CNDP1) on
chromosome 18q. Alleles of this microsatellite encode for a variable number ... Cited by 5 - Related articles - All 2 versions
JM Henderson, MP Alexander, MR Pollak - Journal of the American Society of Nephrology, 2009 - Am Soc Nephrol Mutations in ACTN4, the gene encoding the actin-binding protein -actinin-4, are
a cause of familial FSGS. We examined kidney biopsies from patients with ACTN4
mutations to characterize systematically the histopathology of kidney ... Cited by 2 - Related articles - All 6 versions
FLH Muntinghe, M Verduijn, MW Zuurman, DC … - Journal of the American Society of Nephrology, 2009 - Am Soc Nephrol The CC-chemokine receptor 5 (CCR5) is a receptor for various proinflammatory
chemokines, and a deletion variant of the CCR5 gene (CCR5 32) leads to
deficiency of the receptor. We hypothesized that CCR5 32 modulates ... Cited by 2 - Related articles - All 4 versions
M Murphy, J Crean, D Brazil, D Sadlier, F … - Biochemical Society Transactions, 2008 - biochemsoctrans.org DN (diabetic nephropathy) is the leading cause of end-stage renal disease
worldwide and develops in 25–40% of patients with Type 1 or Type 2 diabetes
mellitus. Elevated blood glucose over long periods together with glomerular ... Cited by 2 - Related articles - Cached - All 4 versions
MP Millis, D Bowen, C Kingsley, RM Watanabe … - Diabetes, 2007 - Am Diabetes Assoc RESULTS— Markers rs13447075 (odds ratio [OR] 1.47 [95% CI 1.14–1.89] per
copy of A allele; P = 0.003) and rs2648862 (2.66 [1.19–5.92] per copy of C
allele; P = 0.008) were strongly associated with ESRD in analyses adjusting ... Cited by 5 - Related articles - BL Direct - All 3 versions
N Papeta, KT Chan, S Prakash, J Martino, K … - The Journal of Clinical Investigation, 2009 - pubmedcentral.nih.gov 1 Department of Medicine, Columbia University College of Physicians and
Surgeons, New York, New York, USA. 2 Department of Genetics, Yale University
School of Medicine, New Haven, Connecticut, USA. 3 Department of Medicine, ... Cited by 2 - Related articles - All 7 versions
JL Anderson, ER Hauser, ER Martin, WK … - Genetic epidemiology, 1999 - ncbi.nlm.nih.gov We performed genome-wide model dependent and independent analyses on a simulated
data set of 400 families segregating for a rare disorder. Regions on chromosomes
1, 3, and 5 were consistently indicated across the various analyses ... Cited by 3 - Related articles - BL Direct
