H Tsui, Y Chan, L Tang, S Winer, RK Cheung, … - Diabetes, 2008 - Am Diabetes Assoc RESULTS— pSC cultures did not express major histocompatibility complex (MHC)
class II and were lysed by ex vivo CTLs from diabetic NOD mice. In vivo,
restoration of MHC class I in GFAP-β2m transgenics significantly ... Cited by 4 - Related articles - BL Direct - All 4 versions
- ►jbc.org AY Savinov, DV Rozanov, AY Strongin - Journal of Biological Chemistry, 2007 - ASBMB Human diabetes mellitus (IDDM; type I diabetes) is a T cell-mediated disease
that is closely modeled in non-obese diabetic (NOD) mice. The pathogenesis of
IDDM involves the transmigration of autoimmune T cells into the pancreatic ... Cited by 3 - Related articles - All 4 versions
EM Zeinstra, N Wilczak, JC Wilschut, L … - Journal of neuroimmunology, 2006 - Elsevier A failure of tight control of MHC class II expression on astrocytes may play a
role in the development of autoimmune responses in multiple sclerosis. The 5-HT
4 serotonin receptor agonists cisapride and prucalopride, at concentrations ... Cited by 4 - Related articles - All 10 versions
CD Sharp, M Huang, J Glawe, DR Patrick, S … - Diabetes, 2008 - Am Diabetes Assoc RESULTS—TNF- significantly increased NOD/LtJ T-cell adhesion, which was
completely blocked by SDF-1 in a dose-dependent manner, revealing a novel
chemorepulsive effect. Conversely, SDF-1 enhanced C57BL/6J T-cell adhesion ... Cited by 4 - Related articles - BL Direct - All 4 versions
I Guleria, M Gubbels Bupp, S Dada, B Fife, Q … - Clinical Immunology, 2007 - Elsevier The PD-1–PDL1 pathway plays a critical role in regulating autoimmune diabetes
as blockade or deficiency of PD-1 or PDL1 results in accelerated disease in NOD
mice. We explored the cellular mechanisms involved in the regulation of ... Cited by 5 - Related articles - All 2 versions
D Yadav, C Fine, M Azuma, N Sarvetnick - Molecular immunology, 2007 - Elsevier Costimulation by B7-1 and B7-2 molecules results in divergent biological
effects. This is particularly striking in the NOD mouse, since the lack of B7-2
leads to complete protection from diabetes whereas the B7-1 deficiency ... Cited by 6 - Related articles - All 3 versions
Y Tsuyuki, H Fujimaki, N Hikawa, K Fujita, T … - Neuroreport, 1998 - journals.lww.com Schwann cells (SCs) are the glial cells that character- ize the peripheral
nervous system (PNS), and corres- pond to oligodendroglia cells in the central
nervous system (CNS). They become aligned along the axonal processes and ... Cited by 15 - Related articles - BL Direct - All 3 versions
- ►cell.com H Tsui, R Razavi, Y Chan, J Yantha, HM … - Trends in Molecular Medicine, 2007 - Elsevier Type 1 diabetes (T1D) results from autoimmune-mediated loss of insulin-producing
β-cells. Recent findings suggest that the events controlling T1D development
are not only immunological, but also neuronal in nature. In the non-obese ... Cited by 8 - Related articles - All 4 versions
- ►diabetesjournals.org J Carrillo, MC Puertas, A Alba, RM Ampudia, X … - Diabetes, 2005 - Am Diabetes Assoc B-cells accumulate in pancreatic islets during the autoimmune response that
precedes the onset of type 1 diabetes. However, the role and antigenic
specificity of these cells remain a mystery. To elucidate the antigenic ... Cited by 14 - Related articles - All 6 versions
