- ►diabetesjournals.org [PDF] S Klinger, C Poussin, MB Debril, W Dolci, PA … - Diabetes, 2007 - Am Diabetes Assoc 1 Institute of Physiology, 2 Center for Integrative Genomics, University of
Lausanne, 1015 Lausanne, Switzerland, 3 Department of Genetic Medicine and
Development, University Medical Center, University Hospital, 1211 Geneva 4, ... Cited by 1 - Related articles - All 2 versions
- ►diabetesjournals.org S Klinger, C Poussin, MB Debril, W Dolci, PA … - Diabetes, 2008 - Am Diabetes Assoc OBJECTIVE—Glucagon-like peptide-1 (GLP-1) is a growth and differentiation
factor for mature β-cells and their precursors. However, the overall effect of
GLP-1 on increasing β-cell mass in both in vivo and in vitro conditions is ... Cited by 9 - Related articles - BL Direct - All 3 versions
B Budowle, JM Roseman, RCP Go, BO Barger … - Diabetologia, 1984 - Springer Summary. The complement component C4 variants C4A4, C4B 4 and C4B Q0 were found
to be significantly increased in 64black patients with Type 1
(insulin-dependent) diabetes compared with 169 black control subjects, ... Cited by 4 - Related articles - All 3 versions
CD Sharp, M Huang, J Glawe, DR Patrick, S … - Diabetes, 2008 - Am Diabetes Assoc RESULTS—TNF- significantly increased NOD/LtJ T-cell adhesion, which was
completely blocked by SDF-1 in a dose-dependent manner, revealing a novel
chemorepulsive effect. Conversely, SDF-1 enhanced C57BL/6J T-cell adhesion ... Cited by 4 - Related articles - BL Direct - All 4 versions
- ►endojournals.org JL Fowlkes, RC Bunn, L Liu, EC Wahl, HN … - Endocrinology, 2008 - Endocrine Soc Type 1 diabetes mellitus is associated with a number of disorders of skeletal
health, conditions that rely, in part, on dynamic bone formation. A mouse model
of distraction osteogenesis was used to study the consequences of ... Cited by 4 - Related articles - BL Direct - All 5 versions
RL Nolsøe, F Pociot, D Novick, M Rubinstein … - Annals of the New York Academy of Sciences, 2003 - ncbi.nlm.nih.gov Type 1 (insulin-dependent) diabetes, T1DM, is the result of an immune-mediated
destruction of the pancreatic beta cells dependent mainly on T helper cells and
macrophages. Interleukin-18 (IL-18) is a proinflammatory cytokine produced ... Cited by 4 - Related articles - BL Direct - All 3 versions
[CITATION] RISC Consortium UK Type 2 Diabetes Genetics Consortium. Common variants of the novel …
- ►pnas.org X Tai, F Van Laethem, AH Sharpe, A Singer - Proceedings of the National Academy of Sciences, 2007 - National Acad Sciences CTLA-4-deficient mice develop a lethal autoimmune lymphoproliferative disorder
that is strictly dependent on in vivo CD28 costimulation. Nevertheless, it is
not known whether there is a specific site on the CD28 molecule that is ... Cited by 4 - Related articles - BL Direct - All 7 versions
- ►clinchem.org S Rani, M Clynes, L O'Driscoll - Clinical Chemistry, 2007 - Am Assoc Clin Chem Methods: We isolated mRNA from medium conditioned by the culture of several
insulin-producing cell types: MIN6(L) (glucose-responsive), MIN6(H)
(glucose-nonresponsive), and MIN6 B1 murine beta cells and monkey kidney ... Cited by 4 - Related articles - BL Direct - All 3 versions
- ►diabetesjournals.org C Meagher, G Arreaza, A Peters, CA Strathdee … - Diabetes, 2007 - Am Diabetes Assoc We previously reported that interleukin (IL)-4 treatment of nonobese diabetic
(NOD) mice elevates intrapancreatic CCL4 expression and protects from type 1
diabetes. Here, we show that antibody neutralization of CCL4 abrogates the ... Cited by 4 - Related articles - BL Direct - All 5 versions
