HJ Schwartz, FC Lowell, JC Melby - Annals of Internal Medicine, 1968 - Am Coll Physicians The eosinopenic response to 40 mg of cortisol given intravenously was studied in
19 unselected asthmatics (group A) and in 6 asthmatics who exhibit clinical
resistance to cortisol's antiasthma effect (group B). The changes in total ... Cited by 86 - Related articles - All 3 versions
- ►nih.gov [PDF] J Carmichael, IC Paterson, P Diaz, GK … - British Medical Journal, 1981 - bmj.com Page 1. doi:10.1136/bmj.282.6274.1419 1981;282;1419-1422 Br Med J (Clin Res Ed)
J Carmichael, IC Paterson, P Diaz, GK Crompton, AB Kay and IW Grant ... Cited by 131 - Related articles - All 7 versions
J Alvarez, W Surs, DY Leung, D Ikle, EW … - The Journal of allergy and clinical immunology, 1992 - ncbi.nlm.nih.gov 1: J Allergy Clin Immunol. 1992 Mar;89(3):714-21. Steroid-resistant asthma:
immunologic and pharmacologic features. Alvarez J, Surs ... Cited by 79 - Related articles - All 2 versions
M Poznansky, A Gordon, J Douglas, A Krajewski … - Clinical Science, 1984 - cs.portlandpress.com In order to investigate the cellular mechanism of glucocorticoid resistance in
chronic asthma, peripheral blood mononuclear cells (MNC) from asthmatic patients
were cultured in soft agar. Cells from patients known to be clinically ... Cited by 56 - Related articles - Cached - All 9 versions
IM Adcock, SJ Lane, CR Brown, MJ Peters, TH … - The Journal of Immunology, 1995 - Am Assoc Immnol Although glucocorticosteroids are a very effective treatment for asthma and
other chronic inflammatory diseases, a small proportion of patients are
resistant to their therapeutic effects. The molecular mechanism for this ... Cited by 119 - Related articles - BL Direct - All 4 versions
CJ Corrigan, PH Brown, NC Barnes, SJ Szefler … - The American review of respiratory disease, 1991 - ncbi.nlm.nih.gov A total of 37 chronic, severe, nonsmoking asthmatic patients with documented
reversible airways obstruction were classified as glucocorticoid-sensitive or
-resistant on the basis of changes in FEV1, FVC, and peak expiratory flow ... Cited by 195 - Related articles - All 2 versions
- ►nih.gov [PDF] AB Kay, P Diaz, J Carmicheal, IW Grant - Clinical and Experimental Immunology, 1981 - pubmedcentral.nih.gov Monocyte complement receptors (MCR) and enhancement of MCR by a monocyte
chemotactic factor (casein) (CRE) were measured in corticosteroid-resistant and
corticosteroid-responsive chronic asthmatics. In newly-diagnosed patients ... Cited by 47 - Related articles - All 2 versions
CJ Corrigan, PH Brown, NC Barnes, JJ Tsai, … - The American review of respiratory disease, 1991 - ncbi.nlm.nih.gov 1: Am Rev Respir Dis. 1991 Nov;144(5):1026-32. Glucocorticoid resistance
in chronic asthma. Peripheral blood T lymphocyte activation ... Cited by 94 - Related articles - All 2 versions
SJ Lane, JP Arm, DZ Staynov, TH Lee - American journal of respiratory cell and molecular …, 1994 - cat.inist.fr Chemical mutational analysis of the human glucocorticoid receptor cDNA
in glucocorticoid-resistant bronchial asthma. SJ LANE, JP ... Cited by 62 - Related articles - BL Direct - All 4 versions
A Vecchiarelli, A Siracusa, E Cenci, M Puliti, G … - Clin Exp Allergy, 1992 - interscience.wiley.com Peripheral blood monocytes (PBM) may be activated in asthmatic patients, a
condition usually reverted by corticosteroid (CS) treatment. In the present
research we have evaluated the spontaneous or lipopolysaccharide ... Cited by 31 - Related articles - All 4 versions
