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Coral Wille
Coral Wille
UAB: The University of Alabama at Birmingham
Verified email at uab.edu
Title
Cited by
Cited by
Year
Differentiation-dependent KLF4 expression promotes lytic Epstein-Barr virus infection in epithelial cells
DM Nawandar, A Wang, K Makielski, D Lee, S Ma, E Barlow, J Reusch, ...
PLoS pathogens 11 (10), e1005195, 2015
932015
Viral genome methylation differentially affects the ability of BZLF1 versus BRLF1 to activate Epstein-Barr virus lytic gene expression and viral replication
CK Wille, DM Nawandar, AR Panfil, MM Ko, SR Hagemeier, SC Kenney
Journal of virology 87 (2), 935-950, 2013
802013
Methyl-metabolite depletion elicits adaptive responses to support heterochromatin stability and epigenetic persistence
SA Haws, D Yu, C Ye, CK Wille, LC Nguyen, KA Krautkramer, ...
Molecular cell 78 (2), 210-223. e8, 2020
532020
Cellular transcription factor Oct-1 interacts with the Epstein-Barr virus BRLF1 protein to promote disruption of viral latency
AR Robinson, SS Kwek, SR Hagemeier, CK Wille, SC Kenney
Journal of virology 85 (17), 8940-8953, 2011
482011
5-hydroxymethylation of the EBV genome regulates the latent to lytic switch
CK Wille, DM Nawandar, AN Henning, S Ma, KM Oetting, D Lee, ...
Proceedings of the National Academy of Sciences 112 (52), E7257-E7265, 2015
322015
Epstein-Barr virus transcription activator R upregulates BARF1 expression by direct binding to its promoter, independent of methylation
EK Hoebe, C Wille, ES Hopmans, AR Robinson, JM Middeldorp, ...
Journal of virology 86 (20), 11322-11332, 2012
272012
Restricted TET2 expression in germinal center type B cells promotes stringent Epstein-Barr virus latency
CK Wille, Y Li, L Rui, EC Johannsen, SC Kenney
Journal of virology 91 (5), 10.1128/jvi. 01987-16, 2017
242017
Epstein–barr virus gene BARF1 expression is regulated by the epithelial differentiation factor ΔNp63α in undifferentiated nasopharyngeal carcinoma
E Hoebe, C Wille, S Hagemeier, S Kenney, A Greijer, J Middeldorp
Cancers 10 (3), 76, 2018
172018
DOT1L inhibition enhances pluripotency beyond acquisition of epithelial identity and without immediate suppression of the somatic transcriptome
CK Wille, R Sridharan
Stem cell reports 17 (2), 384-396, 2022
122022
Connecting the DOTs on cell identity
CK Wille, R Sridharan
Frontiers in Cell and Developmental Biology 10, 906713, 2022
72022
Antagonistic H3K79me-H3K9ac crosstalk determines elongation at housekeeping genes to promote pluripotency
CK Wille, X Zhang, S Haws, J Denu, R Sridharan
bioRxiv, 2022
32022
Dot1L interaction partner AF10 safeguards cell identity during the acquisition of pluripotency
CK Wille, EN Neumann, AJ Deshpande, R Sridharan
BioRxiv, 2020.12. 17.423347, 2020
32020
Dot1L methyltransferase activity is a barrier to acquisition of pluripotency but not transdifferentiation
CK Wille, R Sridharan
bioRxiv, 835413, 2020
32020
DOT1L is a barrier to histone acetylation during reprogramming to pluripotency
CK Wille, X Zhang, SA Haws, JM Denu, R Sridharan
Science Advances 9 (46), eadf3980, 2023
22023
DOT1L interaction partner AF10 controls patterning of H3K79 methylation and RNA polymerase II to maintain cell identity
CK Wille, EN Neumann, AJ Deshpande, R Sridharan
Stem Cell Reports 18 (12), 2451-2463, 2023
2023
Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells
D Greenspan, J Greenspan, DM Nawandar, A Wang, K Makielski, D Lee, ...
2015
Cytosine modification of the Epstein-Barr viral genome affects lytic reactivation and helps enforce latency type
CK Wille
The University of Wisconsin-Madison, 2015
2015
Epstein–Barr Virus Gene BARF1 Expression is Regulated by the Epithelial Differentiation Factor∆ Np63α in Undifferentiated Nasopharyngeal Carcinoma
E Hoebe, C Wille, S Hagemeier, S Kenney, A Greijer, J Middeldorp
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Articles 1–18